Venous stasis occurs when blood flow through the veins is slowed or obstructed, leading to a higher risk of thrombus (blood clot) formation. Stagnant blood is more likely to clot, especially in the lower extremities where gravity makes it harder for blood to return to the heart, increasing the risk of deep vein thrombosis (DVT). Patients with conditions such as obesity, prolonged immobility, or venous insufficiency are at higher risk of developing venous stasis and subsequent thrombus formation.
The nutrient arteries near the epiphyseal cartilage are nonanastomosing, thereby allowing any blockage to produce tissue necrosis and the sinusoids (venous side of capillary) have slow, turbulent flow predisposing to thrombosis.
If a thrombus in the left common iliac vein dislodged, it would likely travel to the pulmonary artery via the venous circulation, as this is the most common site for venous thromboembolism to cause pulmonary embolism.
Syndromes related to venous insufficiency are caused by valve incompetence. Venous insufficiency is a chronic (long term) condition
Normal central venous pressure is in the range of 2-8 cmH2O.
The venous blood from the brain is drained by a network of veins called the cerebral venous sinuses. The largest of these sinuses is the superior sagittal sinus, which ultimately drains into the internal jugular veins.
The nutrient arteries near the epiphyseal cartilage are nonanastomosing, thereby allowing any blockage to produce tissue necrosis and the sinusoids (venous side of capillary) have slow, turbulent flow predisposing to thrombosis.
In medical terms, stasis means lack of movement. For example, venous stasis is a relative lack of flow or slow flow in the veins.
If a thrombus in the left common iliac vein dislodged, it would likely travel to the pulmonary artery via the venous circulation, as this is the most common site for venous thromboembolism to cause pulmonary embolism.
Coagulation or clotting means to stop blood flow.
Arterial thrombosis is associated with an atherosclerotic plaque being ruptured. It is hence associated with high pressure arteries. The thrombus is made of many platelets and contains little fibrin. It can lead to stroke or acute myocardial infarction in the most severe cases. Venous thrombosis is caused by vessel injury or static blood flow in a low-pressure venous system. The thrombus is made of fibrin and red blood cells with very little platelets. It can detach to form an embolus and may cause pulmonary embolism.
Thrombophlebitis is the medical term meaning a clot forms in a vein, and inflammation around the vein results.thrombophlebitis
The abnormal condition of having a thrombus is known as thrombosis. Thrombosis occurs when a blood clot forms in a blood vessel, obstructing blood flow and potentially leading to complications such as embolism or ischemia. It can occur in veins (venous thrombosis) or arteries (arterial thrombosis) and is often associated with risk factors like prolonged immobility, injury, or certain medical conditions.
Elevating an injured leg prevents venous stasis and oedema from forming. Prevention of these processes requires the leg to be mobile to overcome gravity. When your leg is immobile it is important to utilise gravity to prevent these processes from occurring.
Phleboliths are small, round calcifications that can develop in veins, usually in the lower extremities. They are typically benign and result from the gradual deposition of minerals in stagnant blood within the vein. Factors such as venous stasis, inflammation, or trauma can contribute to their formation.
Venous ulcer are caused by venous insufficiency (improper functioning of venous valves).
Wounds occur due to trauma by external means accidental or intentional, by contact or radiation Ulcers occur as a result of an underlying or internal etiology, eg: A venous leg ulcer is the consequence of venous stasis.
Most thrombi originate from areas of turbulence in blood flow, particularly in veins and arteries. In the venous system, they commonly form in deep veins of the legs due to stasis, often associated with conditions like prolonged immobility. In arteries, thrombi frequently develop at sites of atherosclerotic plaques, where endothelial injury and turbulent flow occur. These conditions create an environment conducive to platelet activation and clot formation.