The genetic mutations for junctional EB are found in the genes responsible for producing the protein Laminin-5.
The absence of a P wave on an EKG could indicate an issue with the electrical impulse formation in the atria of the heart, such as in atrial fibrillation where the atria are not depolarizing in a coordinated manner. Other possible causes include atrial flutter, junctional rhythm, or ectopic atrial beats. Further evaluation by a healthcare professional is necessary to determine the underlying cause.
Inflamed junctional mucosa refers to inflammation occurring at the junction where the gums and the lining of the mouth meet. This can be caused by poor oral hygiene, smoking, certain medications, or systemic diseases. Treatment usually involves improving oral hygiene, using anti-inflammatory mouthwash, and addressing any underlying causes.
Junctional folds increase the surface area of cell membranes in tissues like the small intestine, allowing for more efficient absorption of nutrients. They also help in cell-cell communication by providing a larger area for interactions between adjacent cells.
Cell renewal of junctional epithelium takes place at the base of the epithelium through cell division and migration from the basal layer towards the surface. This process occurs in the gingival sulcus, the space between the tooth surface and the epithelial tissue.
EB simplex is typically caused by a mutation in genes that provide instructions for making proteins involved in maintaining the structure of skin cells. These mutations can weaken the attachment of skin layers, leading to blisters and skin fragility. Genes commonly affected include KRT5 and KRT14 which encode keratins that are essential for skin structure.
EB simplex, junctional EB, and dystrophic EB. These can range in severity from mild blistering to more disfiguring and life-threatening disease.
does not lead to scarring. However, skin on the areas prone to blistering, such as elbows and knees, often shrinks
varies depending on the subtype of the disease. Individuals with EB simplex can live long, fulfilling lives. The severity of the junctional and dystrophic forms of EB can vary greatly. Infants affected with some forms of the disease often do not survive
An accelerated junctional rhythm has a heart rate between 60 and 100. Meanwhile, a junctional tachycardia has a heart rate >100.
The junctional epithelium attaches to enamel by means of internal basal lamina .
Yes
The absence of a P wave on an EKG could indicate an issue with the electrical impulse formation in the atria of the heart, such as in atrial fibrillation where the atria are not depolarizing in a coordinated manner. Other possible causes include atrial flutter, junctional rhythm, or ectopic atrial beats. Further evaluation by a healthcare professional is necessary to determine the underlying cause.
Inflamed junctional mucosa refers to inflammation occurring at the junction where the gums and the lining of the mouth meet. This can be caused by poor oral hygiene, smoking, certain medications, or systemic diseases. Treatment usually involves improving oral hygiene, using anti-inflammatory mouthwash, and addressing any underlying causes.
Junctional, confrontational, instructional
Dystrophic EB is caused by mutations in genes for type VII collagen, the protein contained in the fibers anchoring the epidermis to the deeper layers of the skin
P waves occur from the sinoatrial node and indicate the atrial component of a heart rate. Junctional rhythms occur when the AV node (below the sinoatrial node) takes over. Therefore, in most junctional rhythms there are no p waves.
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