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In hyperkalemia, the increased extracellular potassium levels lead to a decrease in the resting membrane potential of cells, making them more excitable. This can result in muscle weakness, cardiac arrhythmias, and potentially cardiac arrest.

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Hyperkalemia causes depolarization of the resting membrane potential, leading to reduced excitability of cells. This shift makes it harder for action potentials to fire, as the threshold for depolarization is increased. Additionally, hyperkalemia can alter the function of voltage-gated sodium channels, further impairing action potential generation.


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