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What happens when binding acetylcholine to the chemically gated ion channels on the plasma membrane of the muscle fiber?
When acetylcholine binds to the chemically gated ion channels on the plasma membrane of the muscle fiber, it causes these channels to open, allowing sodium ions to flow into the cell. This influx of sodium ions depolarizes the muscle fiber membrane, generating an action potential. The action potential then triggers the release of calcium ions from the sarcoplasmic reticulum, ultimately leading to muscle contraction.
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What is a system of membrane channels called?
In the nervous system the system of membrane channels is called the neuroreceptors. Neurotransmitters use the neuroreceptors as binding sites.
What happens to the muscle membrane when acetylcholine binds to receptors at the motor plate?
When acetylcholine binds to receptors at the motor plate, this binding opens ligand-gated ion channels on the motor end plate, ions diffuse through the open ligand gated ion channels, and the flow of ions causes the motor end plate to reach threshold and an action potential is generated at the motor end plate.
When acetylcholine diffuses across the synaptic cleft and binds to receptors on the folded sarcolemma it initaly causes what channels to open in the sarcolemma?
Acetylcholine binding causes nicotinic acetylcholine receptors on the folded sarcolemma to open, allowing the influx of sodium ions into the muscle cell. This initiates an action potential that propagates along the sarcolemma and into the T-tubules, triggering muscle contraction.
What is the open or close in response to binding specific molecules?
In response to binding specific molecules, a receptor can either open or close, depending on its function and the signaling pathways involved. For example, ligand-gated ion channels open in response to binding neurotransmitters, allowing ions to flow across the membrane, while G protein-coupled receptors may trigger intracellular signaling cascades upon ligand binding.
What is the chemical transmitter released at the neuromuscular junction?
Acetylcholine is the primary chemical transmitter released at the neuromuscular junction. It binds to acetylcholine receptors on the muscle cell membrane, leading to muscle contraction.
Binding of the Acetylcholine to receptors causes a by opening what channels that permit both potassium and sodium to permeate the membrane?
Binding of acetylcholine to nicotinic acetylcholine receptors opens ion channels that allow both sodium and potassium ions to permeate the membrane. This causes depolarization of the membrane potential, leading to an excitatory response in the cell.
What neurotransmitter control the somatic nervous system and how does it work?
Acetylcholine (ACh) is the only neurotransmitter used in the motor division of the somatic nervous system. It works by binding to acetylcholine receptors on skeletal muscle fibers and opening ligand-gated sodium channels in the cell membrane.
What neurotransmitter controls the somatic nervous system how does it work?
Acetylcholine (ACh) is the only neurotransmitter used in the motor division of the somatic nervous system. It works by binding to acetylcholine receptors on skeletal muscle fibers and opening ligand-gated sodium channels in the cell membrane.
At the neuromuscular junction, the muscle contraction initiation event is?
binding of acetylcholine to membrane receptors on the sarcolemma
What is a system of membrane channels?
In the nervous system the system of membrane channels is called the neuroreceptors. Neurotransmitters use the neuroreceptors as binding sites.
What part of the muscle cell membrane contain acetylcholine receptors?
Acetylcholine receptors are located on the motor end plate of the muscle cell membrane. This specialized region is where the nerve cell communicates with the muscle cell, allowing for the initiation of muscle contraction in response to acetylcholine binding to its receptors.
What is a system of membrane channels called?
In the nervous system the system of membrane channels is called the neuroreceptors. Neurotransmitters use the neuroreceptors as binding sites.
What happens to the muscle membrane when acetylcholine binds to receptors at the motor plate?
When acetylcholine binds to receptors at the motor plate, this binding opens ligand-gated ion channels on the motor end plate, ions diffuse through the open ligand gated ion channels, and the flow of ions causes the motor end plate to reach threshold and an action potential is generated at the motor end plate.
When acetylcholine diffuses across the synaptic cleft and binds to receptors on the folded sarcolemma it initaly causes what channels to open in the sarcolemma?
Acetylcholine binding causes nicotinic acetylcholine receptors on the folded sarcolemma to open, allowing the influx of sodium ions into the muscle cell. This initiates an action potential that propagates along the sarcolemma and into the T-tubules, triggering muscle contraction.
What is the open or close in response to binding specific molecules?
In response to binding specific molecules, a receptor can either open or close, depending on its function and the signaling pathways involved. For example, ligand-gated ion channels open in response to binding neurotransmitters, allowing ions to flow across the membrane, while G protein-coupled receptors may trigger intracellular signaling cascades upon ligand binding.
Binding of neurotransmitter to the receptors on the motor endplate open?
When a neurotransmitter binds to its receptor on the motor endplate, it triggers the opening of ion channels in the postsynaptic membrane. This allows for the influx of ions, typically leading to depolarization of the muscle cell membrane and initiation of a muscle action potential. Subsequently, this leads to contraction of the muscle fiber.
Name two channels through which chloride ions could pass into the cell?
Chloride ions can pass into the cell through voltage-gated chloride channels and ligand-gated chloride channels. These channels allow for the movement of chloride ions across the cell membrane in response to changes in voltage or binding of specific ligands.
