Large myelinated axons.
The firing rate in a nerve refers to the frequency at which action potentials are generated and conducted along the nerve. This firing rate can vary depending on the type of nerve and the intensity of the stimulus being received.
The sinoatrial (SA) node is innervated by the vagus nerve (parasympathetic) and sympathetic nerves. However, the sympathetic nerves play a more significant role in initiating an impulse in the SA node by releasing norepinephrine, which increases heart rate.
Impulses move slower at lower temperatures because temperature affects the speed of nerve conduction by altering the rate of chemical reactions involved in transmitting the impulse. Cold temperatures slow down these chemical reactions, causing nerve impulses to travel more slowly.
The vagus nerve (cranial nerve X) is responsible for slowing the heart rate (parasympathetic control) and accelerating peristalsis in the gastrointestinal tract.
Vagus nerve carries parasynpathetic nerve fibers .
Optic nerve
The firing rate in a nerve refers to the frequency at which action potentials are generated and conducted along the nerve. This firing rate can vary depending on the type of nerve and the intensity of the stimulus being received.
One factor that does not influence the rate of impulse propagation is the size of the neuron. The rate of impulse propagation is determined mainly by the myelination of the axon, the presence of nodes of Ranvier, and the diameter of the axon.
The sinoatrial (SA) node is innervated by the vagus nerve (parasympathetic) and sympathetic nerves. However, the sympathetic nerves play a more significant role in initiating an impulse in the SA node by releasing norepinephrine, which increases heart rate.
11 mS
Angular impulse is defined as the rate-of-change of the angular acceleration.
The brain can influence the heart in two ways. Sympathetic and parasympathetic stimulation. Parasympathetic stimulation of the heart is through the vagus nerve. Sympathetic stimulation of the heart is via cervical and thoracic splanchnic nerves. The heart has its own pacemaker cells, so the brain only changes the rate and contractility of the heart.
it decreased heart rate , since vagus nerve innervate parasympathetic never and we know parasympathetic nerve reduce heart rate.
Impulses move slower at lower temperatures because temperature affects the speed of nerve conduction by altering the rate of chemical reactions involved in transmitting the impulse. Cold temperatures slow down these chemical reactions, causing nerve impulses to travel more slowly.
An abnormally thin dendritic spine can yield poor neural impulse conduction; and drugs like LSD which mimic neurotransmitters may "clog" receptors & cause continued activation, I think. I suspect there could also be problems with the PRODUCTION of neurotransmitters or with the conveyance of vesicles containing NT to the synapse. Synapses are spaces or junctions between two neurons. The principle of synapse works on mechanism of neuro-transmitters and neuro-inhibitors. When a nerve impulse reaches the end of one neuron to travel to the next one, at that moment acetylcholine is produced in the synapse. Acetyl choline being a neurotransmitter helps to transmit the nerve impulse. Right after the nerve impulse has passed through the synapse, a neuroinhibitor such as cholinesterase si produced to stop the action of acetylcholine. At a particular synapse, these two chemicals are produced at a very high rate. This is how a nerve impulse works. But suppose if the production of these two chemicals is affected, the synapse will not function properly.
Stimulation of the vagus nerve decreases heart rate. The vagus nerve is an important part of the parasympathetic nervous system.
Does the vagus nerve carry impules that increase the heart rate