Neurotransmitters are released into the synaptic cleft when they reach the end of a neuron. From there, they bind to receptors on the postsynaptic neuron, initiating a new electrical signal to continue the communication process. Some neurotransmitters may be recycled back into the presynaptic neuron or broken down by enzymes.
Nerve impulses, which are electrical, do not jump across the synaptic gap at synapses. Instead, the arrival of a nerve impulse at the axon terminal triggers the release of chemicals called neurotransmitters from the axon terminal into the synaptic gap, the nerve impulses then travel across the chemicals to the place where it needs to go to
An electrial nerve impulse travels across a synapse by diffusion. The neurotransmitter substance from the pre-synaptic cleft travels across the synapse via diffusion. This is then received by receptors in the post synaptic cleft
You've sort of got it backwards: axon terminals initially release neurotransmitters into the synaptic cleft, from where they diffuse across the cleft and fit into receptor sites on ligand-gated sodium ion pores on the dendrites, causing those pores to open, allowing sodium ions into the dendrite, resulting in a change in the voltage of the dendrites membrane, which initiates the propagation of the signal along the dendrite and soma towards the axon hillock, where it may trigger an action potential in the axon.However, after the neurotransmitters have done their job at the dendrites, they can be "released" by the dendrites , as in let go of, to be re-absorbed, re-cycled, re-used by the axon terminals.The axons "give" the neurotransmitters to the dendrites as chemical messengers to convey the signal, and the dendrites "give them back" after the message has been received and conveyed onward.
Exitatory neurotransmitters, such as acetylcholine and glutamate, bind as ligands to ligand-gated channel proteins. Once these neurotransmitters have binded to these transport proteins, the channel opens between the outside and inside of the cell. Once open, sodium (Na+) ions tend to rush into the cell from the outside along with the electrochemical gradient, because these ions want to go from high concentration and positive membrane charge to where there is a lower concentration of Na+ and a more negative membrane charge. This action depolarizes the membrane, meaning the difference in voltage between the inside and the outside of the cell membrane becomes less negative. Depolarization of the cell membrane increases the likelihood of firing an action potential down that neuron, opening calcium (Ca2+) channels in the synaptic terminals, causing an influx of calcium, which causes vesicles filled with neurotransmitters to fuse to the presynaptic membrane, releasing the neurotransmitters into the synapse, starting the whole process over again.
The axon terminal is the part of the nerve responsible for sending the message at all. Not only does it send the message, though, it has branching paths which allow it to determine which path it'll go on.
Impulses in the cell body (soma) of a neuron and move on along its axon, which conducts the impulse to a synapse at the end of the axon. There neurotransmitters are released into the synaptic cleft, so that the impulse can stop or go on to the next neuron or a gland/motor end plate, depending on the kind of neurotransmitter. Different types of neurotransmitters are: acetylcholine, adrenaline, noradrenaline, serotonine.
Neurotransmitters are released and go into the synaptic cleft.
Nerve impulses, which are electrical, do not jump across the synaptic gap at synapses. Instead, the arrival of a nerve impulse at the axon terminal triggers the release of chemicals called neurotransmitters from the axon terminal into the synaptic gap, the nerve impulses then travel across the chemicals to the place where it needs to go to
We can be thankful that they go in only one direction; otherwise brain activity would be nothing but chaos. Neurotransmission begins at the synapse. At the synapse, only one of the two corresponding neurons has receptor locations that determine whether or not the receiving neuron will fire. The other neuron at the synapse is responsible for producing the neurotransmitters that attach to the receptors. There is sometimes a re-uptake of neurotransmitters when there are no more receptors for them to attach to. Some psychotropic drugs work to inhibit this re-uptake.
An electrial nerve impulse travels across a synapse by diffusion. The neurotransmitter substance from the pre-synaptic cleft travels across the synapse via diffusion. This is then received by receptors in the post synaptic cleft
Synapses occur between two neurons. Electrical activitiy in the pre-synaptic neuron influences the post-synaptic neuron. There are two types of synapses in the body: Electrical and chemical. Electrical synapses occur in pre and post synaptic neurons that are joined via gap junctions. Currents from action potentials flow across the junction through channels called connexons. This current will depolarize the membrane of the post synaptic neuron to threshold, which will continue the action potential in the cell. Electrical synapses are fast and bidirectional. However, they are mainly found in cardiac and smooth muscles, and not in the mammalian nervous system.Chemical synapses use neurotransmitters. Depolarization occurs in the pre-synaptic neuron and calcium ions rush in. The calcium ions activate neurotransmitter release into the synaptic cleft. The neurotransmitters reach the post-synaptic neuron and cause action potentials to develop.Note: this can go into much more detail
never it can go on for ever
Neurons pass information by neurotransmitters, chemicals that activate receiving neurons. These molecules (neurotransmitters) pass through what is called the synapse: a junction of an axon of the first neuron close to the dendrite of a second neuron. When the neurotransmitters act on the receiving neuron, they can activate a series of changes that cause than neuron to signal to another neuron, resulting in a chain reaction.
You've sort of got it backwards: axon terminals initially release neurotransmitters into the synaptic cleft, from where they diffuse across the cleft and fit into receptor sites on ligand-gated sodium ion pores on the dendrites, causing those pores to open, allowing sodium ions into the dendrite, resulting in a change in the voltage of the dendrites membrane, which initiates the propagation of the signal along the dendrite and soma towards the axon hillock, where it may trigger an action potential in the axon.However, after the neurotransmitters have done their job at the dendrites, they can be "released" by the dendrites , as in let go of, to be re-absorbed, re-cycled, re-used by the axon terminals.The axons "give" the neurotransmitters to the dendrites as chemical messengers to convey the signal, and the dendrites "give them back" after the message has been received and conveyed onward.
go back or get out , its simple
It means its time to get up and go to work.
Exitatory neurotransmitters, such as acetylcholine and glutamate, bind as ligands to ligand-gated channel proteins. Once these neurotransmitters have binded to these transport proteins, the channel opens between the outside and inside of the cell. Once open, sodium (Na+) ions tend to rush into the cell from the outside along with the electrochemical gradient, because these ions want to go from high concentration and positive membrane charge to where there is a lower concentration of Na+ and a more negative membrane charge. This action depolarizes the membrane, meaning the difference in voltage between the inside and the outside of the cell membrane becomes less negative. Depolarization of the cell membrane increases the likelihood of firing an action potential down that neuron, opening calcium (Ca2+) channels in the synaptic terminals, causing an influx of calcium, which causes vesicles filled with neurotransmitters to fuse to the presynaptic membrane, releasing the neurotransmitters into the synapse, starting the whole process over again.