Increasing extracellular potassium concentration can depolarize the cell membrane potential because potassium ions are leaking out of the cell less efficiently, leading to an accumulation of positive charge outside the cell. This disrupts the normal balance of ions and can make it easier for the cell to depolarize and generate an action potential.
A change in extracellular sodium concentration would not alter the resting membrane potential of a neuron because the resting potential is primarily determined by the relative concentrations of sodium and potassium ions inside and outside the cell, as mediated by the sodium-potassium pump and leak channels. Changes in extracellular sodium concentration would not directly affect this equilibrium.
Opening or closing of ion channels at one point in the membrane produces a local change in the membrane potential, which causes electric current to flow rapidly to other points in the membrane.
A sudden increase in membrane potential, typically from a resting membrane potential of around -70mV to a threshold potential of around -55mV, triggers the opening of voltage-gated sodium channels leading to depolarization and initiation of an action potential.
Resting potential and action potential are both names for the measure of electrical voltage within the membrane of a cell. Specifically, these terms are used in describing the transfer of information along neural pathways. Resting potential is a state where cells are at rest. However, if an electrical response or depolarization reaches threshold, then ion channels open, allowing sodium ions to rush into the membrane and increase the voltage measure, firing an action potential along the length of this membrane.
Action potential
A change in extracellular sodium concentration would not alter the resting membrane potential of a neuron because the resting potential is primarily determined by the relative concentrations of sodium and potassium ions inside and outside the cell, as mediated by the sodium-potassium pump and leak channels. Changes in extracellular sodium concentration would not directly affect this equilibrium.
the membrane potential became less negative because less potassium ions went out of the cell (since the extra cellular space has higher concentration of potassium, and potassium goes down its concentration gradient, from high concentration to low concentration). since less potassium (K+)which is positive, left the cell more it became more positive (less negative)
depolarization
despolarization
action potential
Opening or closing of ion channels at one point in the membrane produces a local change in the membrane potential, which causes electric current to flow rapidly to other points in the membrane.
A sudden increase in membrane potential, typically from a resting membrane potential of around -70mV to a threshold potential of around -55mV, triggers the opening of voltage-gated sodium channels leading to depolarization and initiation of an action potential.
The small change in the charge across a neuron's membrane is known as the action potential. It is a brief electrical impulse that travels along the neuron's membrane, allowing for the transmission of signals between neurons.
Action potential
recruitement
Resting potential and action potential are both names for the measure of electrical voltage within the membrane of a cell. Specifically, these terms are used in describing the transfer of information along neural pathways. Resting potential is a state where cells are at rest. However, if an electrical response or depolarization reaches threshold, then ion channels open, allowing sodium ions to rush into the membrane and increase the voltage measure, firing an action potential along the length of this membrane.
During an action potential, the neuron undergoes a rapid change in membrane potential as sodium ions rush into the cell, leading to depolarization. Subsequently, potassium ions move out of the cell, repolarizing the membrane back to its resting state. This rapid change in membrane potential allows for the transmission of electrical signals along the neuron.