No
No
An inferior, not interior, infarction is a subclassification of a heart attack. An inferior myocardial infarction occurs when there is a blockage in the inferior wall of a coronary artery.
An inferior wall mycardial infarction is a heart attack involving the inferior portion of the left ventricle, and in many cases the right ventricle. This is caused by occlusion of the right coronary artery. This can also indicate ischemia (poor oxygenation) of the AV node and bundle of HIS, as these structures are also supplied by the right coronary. An inferior wall MI can be seen in the inferior leads II, III, and AVF.
underactive contracyin of heart muscle
Diaphragmatic attenuation typically refers to a decrease in image quality in the inferior wall of the heart due to interference from the diaphragm. Reversibility in the inferior wall usually indicates that a defect revealed during imaging is potentially reversible, often highlighting areas of reduced blood flow that may improve with treatment.
likely due to coronary artery vasospasm, which can result in transient ischemia. Nitroglycerin dilates coronary arteries, increasing blood flow to the affected area and relieving the spasm-induced wall motion abnormality. It is important to investigate the underlying cause of vasospasm and address any coronary artery disease risk factors.
Give Me a Wall was created in 2005.
the inferior border of the heart is formed by both the right and leftventricles, but the inferior (diaphragmatic) surface of the heart isprimarilymade from the Leftventricle. Kaplen Anatomy 2012, section 3 gross anatomy, page 212.
Rectus abdominis & medial part of the anterolateral abdominal wall
A patient with an inferior wall myocardial infarction (MI) is at risk for several complications, including arrhythmias, particularly bradycardia and atrioventricular (AV) block, due to potential involvement of the right coronary artery and its branches. Additionally, these patients may experience heart failure or cardiogenic shock, especially if there is significant myocardial damage or if the MI is extensive. There is also a risk of recurrent ischemic events and complications related to heart function. Monitoring and prompt intervention are crucial in managing these risks.
Morphine decreases pre-load and after-load and because the Inferior wall is effected the right ventricle is effected. If you do not have enough blood entering the right ventricle when the area is necrotic to begin with you will not be pumping enough blood to perfuse sufficiently. In addition the decreased after-load in in conjunction with the decreased blood pressure caused by the failure of the right ventricle there will not be enough "back-flow" in the cardiac vascular system. In summation if can cause bottoming out of a patient's blood pressure and further cardiac hypo-perfusion. In some cases it has been shown to work if a normal saline or ringers lactate bolus is infused prior to morphine administration.
Severely reduced perfusion in the inferior wall of the heart typically indicates compromised blood flow to that area, often due to coronary artery disease or blockage in the coronary arteries supplying the inferior wall, such as the right coronary artery. This can lead to ischemia, resulting in symptoms like chest pain or heart failure. If left untreated, it may progress to myocardial infarction (heart attack), damaging the heart muscle. Timely medical intervention is crucial to restore blood flow and prevent further complications.