By using a pedigree.

men

2

The difference in longevity between normal people and people with albinism is not well researched. Scientists believe their longevity to be comparable to that of normal people.

Virtually every species of animal has a rare recessive gene mutation that causes the inability to make pigmentation. Their eyes are often very sensitive to sunlight, and they run risk of skin disorders generally from absorbing too much sunlight.

An albino lion would be almost pure white, or perhaps a little pink.

There apparently is also a gene for a white lion due to a recessive gene called: chinchilla or color inhibitor. These aren't considered true albinos. White isn't a particularly good color for lions to be stealthy... but humans have attempted to increase the numbers of individuals expressing these genes.

Albinism doesn't affect a person's life expectancy. People with albinism may be limited in their activities because they can't take exposure to the sun, but their overall lifespan should remain unchanged.

No, not at all. Though because of the light skin and sensitivity to sunlight, albinos are at a larger risk of skin cancer.

yes, of course!! people with aids/cancer/progeria/diabetes can dye their hair. right?

EDIT***

Actually, no, they cannot. When someone is Albino, their hair has no pigment, which means that holding and locking in color is extremely difficult.

ANSWER:

YES!!!! i am albino, i have done it before. but let me say it is a looooot harder to do than color hair but it is a longer processes

ANSWER:

I really wouldn't! I'm a teenager with albinism and with my experience in the salon today I'm never going near hair dye ever again! Originally, i was going to get low lgihts. I had a test strand done a few weeks earlier and it was fine. However I changed my mind and we decided to do a glaze instead of a dye because eventually they said my roots would turn grey. They started putting this nice golden blonde color in my hair and we literally watched my hair turn GREY in minutes ! They rushed me to the sink to get it out. They managed to get most of it out but my hair doesn't look the same and it won't for a few weeks. I really wouldn't do this if you aren't 100% positive your hair will take the color correctly.

ANSWER:

I am 43 year old albino and have been getting my hair colored since I was 18. The very first time the lady butchered it and turned my hair orange. But that was a very long time ago. The place I have been going to over the past 15 years have been fantastic. The first product that they used to put on burned really bad, but was necessary. Now the product hardly burns at all. You simply have to find a place that is willing to keep trying until they get it right. Ask first if they will, if not go elsewhere. Its your money, and its their job to satisfy what you want.

About this Yes people with Albinisme can dye their hair with Hena my color is just brown/redish so that you know that

Yes. They tend to have pale blonde or yellow hair, pink eyes, pink skin, and usually have eye problems. This can occur in any race.

The lack of pigment in the eyes of people with albinism changes the light index of the lens which leads to refractive issues such as extreme farsightedness. They may also have problems with glare.

yes it can be but that is not always the case albinism is a genetic disorder that can just happen for no particular reason like a baby being born with an organ failure or a missing arm or whatever it is not allways passed down from the parents and it can skip gnerations

hope this helped

life is a little harder when you have to have assitance to find what to wear and to compare things.

From all I have read and studied (I am a 2nd semester senior nursing student at a major university) Spina Bifida is not so much a chromosomal defect as a neural defect caused by a lack of folic acid during the very early stages of pregnancy.

This is why women who wish to get pregnant are encouraged to begin taking folic acid BEFORE they actually get pregnant.

One would define albinism as either a complete or partial absence of skin, hair, and eye pigment. This is due to the deficiency of an enzyme called tyrosinase, which is involved in the production of melanin.

It is passed on from mother or father to child if the parents are albino, or if the mother carries the gene for albinism. The father cannot carry the gene as only woman are able to be carriers. Therefore, even if both parents are normal, the child can be albino (because the mother obviously carries the gene for albinism).

:D

Albinism is a genetic defect where the person afflicted is homozygous recessive for albinism. So it cannot be cured.

Topical treatment involves wearing clothing or sunblock to avoid skin damage, as there is usually not enough melanin to tan the skin. Protecion of the eyes, which also lack pigment in the iris, is also a concern. This can lead to retinal damage in some individuals, and should be monitored.

1 in 17,000, or 0.0059%

1 in 70, or 1.4% are carriers.

The estimated 2007 population of the US is 302,854,000

Albinism's effects vary with its severity, with outcomes ranging from combined pigment loss in the hair, skin and iris to pigment loss limited to the retina.

Discontinuous. There's no middle ground, someone either has it or doesn't.

No pigments in the body. The skin is white and the eyes are red.

http://en.wikipedia.org/wiki/Albinism Albino" redirects here. For other uses, see Albino (disambiguation). Semi-protected Albinism Classification and external resources Girl from Honduras with probable OCA1a-type albinism. ICD-10 E70.3 ICD-9 270.2 OMIM 203100 103470, 203200, 203280, 203290, 203300, 203310, 256710, 278400, 214450, 214500, 220900, 300500, 300600, 300650, 300700, 600501, 604228, 606574, 606952, 607624, 609227 DiseasesDB 318 MedlinePlus 001479 eMedicine derm/12 MeSH D000417 Albinism (from Latin albus, "white"; see extended etymology, also called achromia, achromasia, or achromatosis; not to be confused with albedo) is a form of hypopigmentary congenital disorder, characterized by a partial (in hypomelanism, also known as hypomelanosis) or total (amelanism or amelanosis) lack of melanin pigment in the eyes, skin and hair (or more rarely the eyes alone). Albinism results from inheritance of recessive alleles. The condition is known to affect mammals (including humans), fish, birds, reptiles and amphibians. While the most common term for an organism affected by albinism is "albino" (noun and adjective), the word is sometimes used in derogatory ways towards people; more neutral terms are "albinistic" (adjective) and "person with albinism" (noun). Additional clinical adjectives sometimes used to refer to animals are "albinoid" and "albinic". Albinism is hereditary; it is not an infectious disease and cannot be transmitted through contact, blood transfusions, or other vectors. The principal gene which results in albinism prevents the body from making the usual amounts of the pigment melanin. Most forms of albinism are the result of the biological inheritance of genetically recessive alleles (genes) passed from both parents of an individual, though some rare forms are inherited from only one parent. There are other genetic mutations which are proven to be associated with albinism. All alterations, however, lead to changes in melanin production in the body.[1][2] Albinism was formerly categorized as tyrosinase-positive or -negative. In cases of tyrosinase-positive albinism, the enzyme tyrosinase is present. The melanocytes (pigment cells) are unable to produce melanin for any one of a variety of reasons that do not directly involve the tyrosinase enzyme. In tyrosinase-negative cases, either the tyrosinase enzyme is not produced or a nonfunctional version is produced. This classification has been rendered obsolete by recent research.[3] The chance of offspring with albinism resulting from the pairing of an organism with albinism and one without albinism is low, as discussed in more detail below. However, because organisms can be carriers of genes for albinism without exhibiting any traits, albinistic offspring can be produced by two non-albinistic parents. Albinism usually occurs with equal frequency in both genders.[1] An exception to this is ocular albinism, because it is passed on to offspring through X-linked inheritance. Thus, males more frequently have ocular albinism as they do not have a second X chromosome.[3] Albino Bennett's Wallaby, Bruny Island, Tasmania, Australia Because organisms with albinism have skin that lacks (sufficiently or entirely) the dark pigment melanin, which helps protect the skin from ultraviolet radiation coming from the sun, they can sunburn easily from overexposure. (See human skin color for more information). Lack of melanin in the eye also results in problems with vision, related and unrelated to photosensitivity, which are discussed further below. Most humans and many animals with albinism appear white or very pale; the multiple types of melanin pigment are responsible for brown, black, gray, and some yellow colorations. In some animals, especially albinistic birds and reptiles, ruddy and yellow hues or other colors may be present on the entire body or in patches (as is common among pigeons), due to the presence of other pigments unaffected by albinism such as porphyrins, pteridines and psittacins, as well as carotenoid pigments derived from the diet. Some animals are white or pale due to chromatophore (pigment cell) defects, do not lack melanin production, and have normal eyes; they are referred to as leucistic. The direct opposite of albinism, an unusually high level of melanin pigmentation (and sometimes absence of other types of pigment in species that have more than one), is known as melanism, and results in an appearance darker than non-melanistic specimens from the same genepool.[4] Albinism-like conditions may affect other pigments or pigment-production mechanisms in some animals (e.g. "whiteface", a lack of psittacins that can affect some parrot species.).[5] Another is common in reptiles and amphibians: axanthism, in which xanthophore metabolism, instead of synthesis of melanin, is affected, resuling in reduction or absence of red and yellow pteridine pigments.[6] Of all these conditions, only albinism and melanism affect humans. An albino Wistar rat, a strain commonly used for both biomedical and basic research. The eyes of an animal with albinism occasionally appear red due to the underlying retinal blood vessels showing through where there is not enough pigment to cover them. In humans this is rarely the case, as a human eye is quite large and thus produces enough pigment to lend opacity to the eye, often colouring the iris pale blue. However, there are cases in which the eyes of an albinistic person appear red or purple, depending on the amount of pigment present. The albinistic are generally (but see related disorders below) as healthy as the rest of their species, with growth and development occurring as normal, and albinism by itself does not cause mortality[1] (though the lack of pigment is an elevated risk for skin cancer and other problems.) Many animals with albinism lack their protective camouflage and are unable to conceal themselves from their predators or prey; the survival rate of animals with albinism in the wild is usually quite low.[7][8] However the novelty of albino animals has occasionally led to their protection by groups such as the Albino Squirrel Preservation Society. Intentionally-bred albinistic strains of some animal species are commonly used as model organisms in biomedical study and experimentation. Examples include the BALB/c mouse and Wistar and Sprague Dawley rat strains, while albino rabbits were historically used for Draize toxicity testing. [9] Albino axolotls, zebrafish, medaka and frogs are other common laboratory animals. The yellow mutation in fruit flies is their version of albinism. The incidence of albinism can be artificially increased in fish by exposing the eggs to heavy metals.[10] About 1 in 17,000 human beings has some type of albinism, although up to 1 in 70 is a carrier of albinism genes.[11]