Ebola Hemorrhagic Fever

An N95 mask can provide some level of protection against the Ebola virus by filtering out large respiratory droplets that may contain the virus. However, it is not considered sufficient on its own for full protection, as Ebola is primarily transmitted through direct contact with infected bodily fluids rather than airborne transmission. For optimal protection, additional personal protective equipment (PPE), such as face shields, gowns, and gloves, is recommended in healthcare settings.

The Ebola virus is primarily found in Central and West Africa. Countries such as the Democratic Republic of the Congo, Guinea, Liberia, Sierra Leone, and Nigeria have reported outbreaks. The virus is transmitted through direct contact with bodily fluids of infected individuals or animals, such as fruit bats and primates. Efforts to control outbreaks focus on public health measures, community engagement, and vaccination.

Ebola virus can have severe effects on pregnant women, often leading to complications such as miscarriage, stillbirth, or maternal death. The virus can cross the placenta, posing risks to the fetus and potentially leading to vertical transmission. Additionally, pregnant women with Ebola may experience more severe symptoms compared to non-pregnant individuals, complicating treatment and recovery. The overall impact highlights the urgent need for targeted healthcare strategies for pregnant women during Ebola outbreaks.

Ebola is caused by the Ebola virus, which is transmitted to humans through contact with infected animals, such as fruit bats or primates, and spreads among people through bodily fluids. Prevention strategies include practicing good hygiene, using personal protective equipment (PPE) in healthcare settings, and avoiding contact with infected individuals or animals. Vaccination, such as the rVSV-ZEBOV vaccine, has been effective in outbreak control. Additionally, public health education and rapid response to outbreaks are crucial in managing and preventing the spread of Ebola.

No, Ebola is not a monera type. Monera refers to a kingdom that includes prokaryotic organisms such as bacteria and archaea. Ebola is a virus, specifically belonging to the Filoviridae family, which is classified as a part of the viral domain rather than the monera kingdom.

Filoviruses are a family of viruses known for their thread-like appearance and ability to cause severe hemorrhagic fevers in humans and other primates. The most well-known members of this family include the Ebola virus and the Marburg virus, both of which can lead to high mortality rates. Filoviruses are typically transmitted through direct contact with infected bodily fluids or contaminated surfaces. Research into these viruses is crucial for developing effective treatments and vaccines.

The United States experienced an outbreak of Ebola in 2014, primarily linked to the West African Ebola epidemic. The first confirmed case in the U.S. was reported in September 2014, involving a man who traveled from Liberia to Dallas, Texas. Subsequent cases included healthcare workers who treated him, leading to heightened awareness and response efforts.

Antes de entrar em uma célula, o vírus Ebola precisa passar por algumas etapas essenciais para o processo de infecção:

1. Ligação aos receptores celulares: O vírus Ebola possui proteínas de superfície, chamadas glicoproteínas, que se ligam a receptores específicos na membrana da célula hospedeira. Um desses receptores é a NPC1 (Niemann-Pick C1), encontrado em células como macrófagos, células dendríticas e células endoteliais.

2. Internalização: Após a ligação aos receptores, o vírus é englobado pela célula por um processo chamado endocitose, onde ele é "engolido" e levado para dentro da célula em uma vesícula.

3. Fusão e liberação do material genético: Dentro da célula, o vírus precisa que a vesícula se funda com a membrana viral, liberando seu material genético no citoplasma para começar a se replicar.

Essas etapas são fundamentais para o Ebola infectar e se multiplicar nas células hospedeiras.

No lol

The current school of thought is that bats, which harbor a large number of zoonotic viruses (ones that can be passsed from one species to another) without ill effects to the bat are the primary source of infection in the current

out break. Other species of animals can transmit the virus as well, however, they are considered to be short term vectors as the disease is also terminal

for them as well.

Currently the WHO (World Health Organization) is blaming the outbreak on the consumption of bats and a number of countries have banned the sale and consumption of bats.

There is ample evidence that ebola is a naturally-occurring virus. There are many animals with antibodies to ebola and similar viruses. It is human nature to look for a conspiracy to explain such a natural force, but there is no science behind the belief that ebola is man-made.

cIDQUE

People aren't able to rise from the dead. The Ebola victims were mistakenly thought to be dead but were not.

You should know that Jack and Brendans Webshow knows. Youtube them!

Isolation

There are no medical cures and the death rate is high. Some speculate that those who have survived ebola did so because their own immune systems fought off the disease. At this point, it seems having a healthy immune system is the best defense against the disease. Some also claim taking Vitamin C in healthy doses, IV Vitamin C, or NanoSilver might increase chances of survival, but that has not been confirmed.

Ebola would be an epidemic. The difference between a pandemic and an epidemic is the population size and locality of infection. An epidemic is a greater than normal amount of infection in a particular area or when infection occurs in an area that isn't normally associated with a certain disease. A pandemic is when the epidemic reaches to world wide proportions.

Symptoms start two days to three weeks after contracting the virus, with a fever, sore throat, muscle pain, and headaches. Typically nausea, vomiting, and diarrhea follow, along with decreased functioning of the liver and kidneys.
Around this time, affected people begin to bleed both within the body and externally. Prolonged cases are often complicated by the occurrence of long-term problems, such as inflammation of the testicles, joint pains, muscle pains, skin peeling, or hair loss. Eye symptoms, such as light sensitivity, excess tearing, iritis, iridocyclitis, choroiditis, and blindness have also been described.

No, fruit bats are in fact hervibores and post no physical threat to humans and animals. However, it should be noted that some bats have been known to carry diseases that are known to be dangerous to humans.

One lab study found that Ebola could live for up to 6 days under ideal conditions. But it wouldn’t likely last that long in most places. Household bleach can kill it. U.S. hospitals are so good about cleaning and disinfecting that experts believe the virus could last about 24 hours, at most.

Ebola has been around 38 years.

No.

Of course it is. Watch out for the soft kill vaccine on the back of it. Bound to contain the carcinogen formaldehyde, like the Pandemrix Swine Flu hoax non-vaccine, which was withdrawn due to multiple cases of narcolepsy.

no it was created by a monkey

The Ebola virus was started by contaminated water.

The fusion of the rabies virus envelope to the host cell membrane (adsorption) initiates the infection process. The interaction of the G protein and specific cell surface receptors may be involved. After adsorption, the virus penetrates the host cell and enters the cytoplasm by pinocytosis (via clathrin-coated pits). The virions aggregate in the large endosomes (cytoplasmic vesicles). The viral membranes fuse to the endosomal membranes, causing the release of viral RNP into the cytoplasm (uncoating). Because lyssaviruses have a linear single-negative-stranded ribonucleic acid (RNA) genome, messenger RNAs (mRNAs) must be transcribed to permit virus replication. A viral-encoded polymerase (L gene) transcribes the genomic strand of rabies RNA into leader RNA and five capped and polyadenylated mRNAs, which are translated into proteins. Translation, which involves the synthesis of the N, P, M, G and L proteins, occurs on free ribosomes in the cytoplasm. Although G protein synthesis is initiated on free ribosomes, completion of synthesis and glycosylation (processing of the glycoprotein), occurs in the endoplamsic reticulum (ER) and Golgi apparatus. The intracellular ratio of leader RNA to N protein regulates the switch from transcription to replication. When this switch is activated, replication of the viral genome begins. The first step in viral replication is synthesis of full-length copies (postive strands) of the viral genome. When the switch to replication occurs, RNA transcription becomes "non-stop" and stop codons are ignored. The viral polymerase enters a single site on the 3' end of the genome, and proceeds to synthesize full-length copies of the genome. These positive strands of rabies RNA serve as templates for synthesis of full-length negative strands of the viral genome. During the assembly process, the N-P-L complex encapsulates negative-stranded genomic RNA to form the RNP core, and the M protein forms a capsule, or matrix, around the RNP. The RNP-M complex migrates to an area of the plasma membrane containing glycoprotein inserts, and the M-protein initiates coiling. The M-RNP complex binds with the glycoprotein, and the completed virus buds from the plasma membrane. Within the central nervous system (CNS), there is preferential viral budding from plasma membranes. Conversely, virus in the salivary glands buds primarily from the cell membrane into the acinar lumen. Viral budding into the salivary gland and virus-induced aggressive biting-behavior in the host animal maximize chances of viral infection of a new host.