Bone resorption is promoted by the parathyroid hormone, also called PTH. Bone resorption is the process by which osteoclasts break down bone and release the minerals, resulting in a transfer of calcium from bone fluid to the blood.
An increase in bone growth is promoted by different hormones. In most cases this is as a result of estrogen in women and testosterone in the men.
A cell called an osteoclast accomplishes bone resorption.
The osteoclasts carry out bone resorption. In contrast, the osteoblasts create new bone.
In general, it increases calcium in the blood by increasing bone resorption. The exception is when pth is given in spiked pulses, which is the case in some medications (forteo, a pth analogue) which can paradoxically decrease bone resorption.
Hyperparathyroidism or over production of parathyroid hormone causes pain in bones and pathological fractures of bones. Due to this problem, you have increased bone resorption and so that is the result of the same.
Resorption is the process of dissolving bone and returning its minerals to the bloodstream.
Osteoclasts
Subperiosteal bone resorption of the distal phalanges means that the bone at the ends of the fingers is disappearing. It's typically a sign of hyperparathyroidism.
Resorption
When osteoclasts break down bone that is called resorption.
Bone resorbing cells are known as osteoclasts. Immature bone cells are referred to as osteoblasts. Mature bone cells are called osteocytes.
PTH is secreted by the parathyroid glands (which are attached to your thyroid) and acts on bone (via increased bone resorption) , the kidneys (via increased reabsorption of calcium) and the intestine (increased calcium absorption by increased levels of activated vitamin D) to increase blood calcium levels.