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IP3. Inositol triphosphate. Cut from the membrane by something I can not remember ( a G protein action ) and then this lipid as part of this signal trasduction pathway docks on the ER and Ca(2+) is released to preform the cellular response.
Wiki User
∙ 12y ago
Wiki User
∙ 11y agoIP3 Inositol triphosphate.
Cleaved from it's position in the cell membrane it docks on the ER and releases Ca 2+.
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Why don't prokaryotes have endoplasmic reticulum?
they don't have organelles, since the cytoplasm does the metabolic work, and technically we will only find the circular DNA in the nucleoid region and some ribosomes (which are NOT organelles, 'cause an organelle is a cellular structure that is protected by a membrane, like mitochondria, chloroplasts) in a prokaryotic cytoplasm.
Why is cox enzyme attached to inner surface of endoplasmic recticulum?
cause it is nice there init
What would happen if acetylcholine was released into a neuromuscular junction?
It attaches to its receptor, and binds. Causes another action potential, (calcium released) and it goes to the T-tubule of the Sarcoplasmic reticulum, and attaches to troponin, which moves the tropomyosin, so the myosin and actin and attach, and cause a muscle contraction.
What cause Calcium in eye?
There are several different conditions that can cause calcium deposits in the eye. These include hypocalcemia, gout, renal failure, and chronic inflammation.
How does plain calcium taste?
do not take pure calcium after a period of time it will cause serious health risks
Why is the endoplasmic reticulum rough?
Because it is studded with ribosomes that cause it to be rough.
Which cell structures cause the outer surface of the rough endoplasmic reticulum to appear rough?
The organelles that give the rough endoplasmic reticulum its rough appearance are ribosomes. When there are no ribosomes present it is called a smooth endoplasmic reticulum.
What would be the effects of adding the sequence KDEL to insulin?
KDEL an amino acid sequence which signals that a protein belongs in the endoplasmic reticulum. Attaching it to insulin would cause the insulin to be retained within the endoplasmic reticulum.
If Calcium concentrations were low muscle contractions might fail because?
It is the release of calcium from the sarcoplasmic reticulum that then binds with actin-myosin ATPase to cause contractions. Low calcium would first affect the release of acetylcholine from the pre-synaptic terminus.
What does sarcoplasmic reticulum do?
The Sarcoplasmic Retiulum releases calcium ions that will cause troponin/tropomyosin complex to move. This exposes the binding sites on actin and allows the cross-bridges of myosin to bind to the actin binding sites.
Normally stored in the terminal cisternae of the sarcoplasmic reticulum?
One major function of the sarcoplasmic reticulum is the regulate the intracellular levels of CA2+ for contractions and relaxation for muscles. Sarcoplasmic Reticulum is a storage organelle with calcium ion pumps on membrane and use ATP to fuel pumps to get calcium ion inside it's cell and holds it. When contraction of muscle is needed calcium ion is then dumped into cytoplasm to cause contraction.
When is calcium pumped into the sarcoplasmic reticulum?
When Ach enters the synaptic cleft (gap) it open chemically gated sodium channels that starts an action potential spreading through the sarcolemma of the myofibril. This action potential spreads down the T-tubules and "shocks" the sarcoplasmic reticulum into releasing calcium ions.
Why don't prokaryotes have endoplasmic reticulum?
they don't have organelles, since the cytoplasm does the metabolic work, and technically we will only find the circular DNA in the nucleoid region and some ribosomes (which are NOT organelles, 'cause an organelle is a cellular structure that is protected by a membrane, like mitochondria, chloroplasts) in a prokaryotic cytoplasm.
How does the nerve impulse cause contraction of the muscle?
The nerve impulse causes the release of acetylcholine from the motor end plate. This causes the depolarization of the membrane of the adjacent muscle cell. Depolarization triggers the release of calcium ions from the sarcoplasmic reticulum inside the muscle cell. In the presence of ATP, the high calcium level causes the myosin heads to bend, dragging actin filaments towards the middle of the unit of contraction.
What happens when calcium ions bind to troponin?
When Ca2+ ions are released from the sarcoplasmic reticulum, They combine with troponin, and this cause the tropomyosin threads to shift their position
Can muscle tissue secrete?
As a matter of fact it can. In the muscle fibers there is a body called the Sarcoplasmic Reticulum which releases calcium ions into your system. These cause a difference in charge resulting in chemical reactions which causes your muscles to contract.
What is contraction and relaxation?
Nerve impulse, or electrical signal, travels down the nerve to the terminal to cause the release of the neurotransmitter acetylcholine (ACh).ACh diffuses across the neuromuscular junction and binds to the receptor sites.Stimulation of the receptor sites causes an electrical impulse to form in the muscle membrane. The electrical impulse travels along the muscle membrane and penetrates deep into the muscle through the T-tubular system.The electrical impulse stimulates the sarcoplasmic reticulum to release calcium into the sarcomere (a contractile unit of a mofibril) area.Calcium allows the actin, myosin, and ATP to interact, causing crossbridge formation and muscle contraction. This process continues as long as calcium is available to the actin and myosin.Muscle relaxation occurs when calcium is pumped back into the sarcoplasmic reticulum, away from the actin and myosin. When calcium moves in this way, the actin and myosin cannot interact, and the muscle relaxes.
