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Compare how a substance that blocks acetylcholinesterase affects neural transmission to a substance that blocks sodium ion channels?
Marlene1976
I had this question in my A&P class. Here is how I answered it:
Acetylcholinesterase or AChE, is the enzyme that breaks down acetylcholine into two separate entities, acetate and choline (Martini, 2006, p. 407). This is the fourth and final step in the cholinergenic synapse. AChE is in the postsynaptic membrane and in the synaptic cleft. This separation makes it easy for the choline to be absorbed into the synaptic bulb which is used for the synthetic production of more acetylcholine along with the use of acetate that is furnished by CoA or coenzyme A.
The affect that this separation has on neural transmissions in blocking sodium ion channels is for the creation of an action potential. Without an action potential, one would not be able to have a muscle contraction and therefore, would not be able to move, if I understand this correctly. Please, correct me if I am wrong.
A hyperpolarization must happen for the sodium ions channels to be blocked. This event is because of IPSP or an inhibitory post synaptic potential (Martini, 2006, p. 412). When this happens, there is a lessened chance for an action potential to be generated.
References:
Martini, F. (2006). Fundamentals of Anatomy & Physiology. San Francisco, CA: Pearson Benjamin Cummings.
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