Synapse
You could locate sites involved in transmitting a nerve impulse by finding the synapses. They are defined as a junction between two nerve cells, consisting of a minute gap across which impulses pass by diffusion of a neurotransmitter.
Synapse? Dendrite? Dendritic spine? Or, "You could maybe a better answer to your question get if you re-stated it less confusingly as?" ie, perhaps, "Where are the receptor sites involved in transmitting a nerve impulse LOCATED?"
H2 refers to nerve cells in your body and brain which are able to receive Histamines. There are H1 and H2 receptors for Histamines, maybe others. So, an H2 antagonist will prevent Histamines from being absorbed by the nerve cell receptor sites.
Neurotransmitter receptor sites on ligand-gated ion pores.
endorphins
An adrenergic receptor is any of several sites in the surface membranes of cells innervated by adrenergic neurons.
dendrites only
endorphine
are located in the same areas of the hypothalamus.
Rapid administration of an antidote can help prevent or mitigate the consequences of some nerve agents. Some nerve agents such as organophosphates can be managed with 2-pam Chloride and Valium. The 2-pam Chloride blocks receptor sites preventing the organophosphate from binding to receptor sites and Valium aids in the prevention or termination of seizures. in organophosphate poisoning, the greatest risk of brain damage comes from the body's inability to effectively breathe during seizures, or effectively exchange oxygen in the alveoli of the lungs due to excessive fluid and mucus production. Essentially, a lack of oxygen causes neurological damage.
Rapid administration of an antidote can help prevent or mitigate the consequences of some nerve agents. Some nerve agents such as organophosphates can be managed with 2-pam Chloride and Valium. The 2-pam Chloride blocks receptor sites preventing the organophosphate from binding to receptor sites and Valium aids in the prevention or termination of seizures. in organophosphate poisoning, the greatest risk of brain damage comes from the body's inability to effectively breathe during seizures, or effectively exchange oxygen in the alveoli of the lungs due to excessive fluid and mucus production. Essentially, a lack of oxygen causes neurological damage.
Since only 1 neurotransmitter fits in the receptor sites, you can say that the receptor sites is the locked door waiting for the neurotransmitter "keys".