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How does a neuron restore a membrane potential following the generation of an action potential?
Wiki User
∙ 10y ago
The hyperpolarization of the membrane potential relative to the resting potential (the undershoot) causes voltage-dependent Potassium conductance (and any Sodium channels not yet inactivated) to turn off, allowing the membrane potential to return to resting level.
Wiki User
∙ 14y ago
Wiki User
∙ 15y agoOnce the action potential has passed there are alot of K ions outside the cell and a alot of Na ions inside the cells. This would not allow another AP potential to pass as there is no concentration gradients for the ions to move down. To return the membrane to its resting potential an enzyme called sodium potassium ATPase used energy from ATP to pump 3 sodium out and 2 potassium in at the same time. This returns the concentration gradient and thanks to the difference in the number of each ion moved it also restores the electrical gradient of "membrane potnetial"
Wiki User
∙ 10y agoOpening the k channels
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What is a sequence of rapidly occurring events that decrease and reverse the membrane potential and then eventually restore it to the resting state?
Action potential or impulses
Describe the events occurring in the cell membrane that permit the conduction of an impulse?
When a nerve impulse is conducted, the neuronal cell membrane undergoes changes in electrical potential. This starts with a rapid influx of sodium ions into the cell through voltage-gated sodium channels, depolarizing the membrane. This depolarization triggers the opening of adjacent sodium channels, resulting in an action potential that travels along the membrane. After the impulse passes, the sodium channels close, and potassium channels open, allowing potassium ions to exit the cell and restore the resting potential.
How is resting potential achieved?
The resting membrane potential difference between the inside and the outside of the cell is the result of selective permeability of the cell membrane and the active transport of ions into and out of the cell. Almost all cells have a potential difference, but some cells, neuron and heart muscle, also have voltage and chemically gated channels that allow for transient deviations from the resting potential.
What happens when action potential reaches presynaptic terminal?
Action potentials propagate from an influx of Na and an efflux of K along an excitable cell (neuronal or muscular). If you think of a zipper with two heads attached to the top, as one zipper head traverses down and opens the zipper the next zipper goes down to close. The first zipper head is the action potential going down an axon. It is able to proceed because there is a membrane potential difference between outside the cell and inside the cell. A normal neuron has a membrane potential of -70mV. That means inside the cell is more negative than outside the cell. So when an action potential is elicited, Na rushes in and K rushes out. This produces slight changes in the membrane potential causing it to go up to around +35mV (inside cell). As this happens right next to that Na and K channels are more Na and K channels that see this happening and they open up in response. This occurs like the first zipper head going down. The second zipper going down is the efflux of Na and influx of K to restore the membrane potential back to normal. When the action potential reaches the end, called terminal bouton, calcium channels that are there waiting for this action potential open up and allow a rush of calcium into the terminal bouton. The calcium serves a separate function to push out little vesicles called neurotransmitters out of the cell to continue an action potential into a different cell.
How does the sodium and potassium pump act to maintain the resting membrane potential?
Primary active transport is the process in which ions are moved across cell membranes against the electrochemical gradient using energy supplied directly be ATP. The action of the sodium-potassium pump is an important example of primary active transport.Secondary active transport is indirectly driven by primary transport. In the sodium-potassium pump, by pumping against the gradient, energy is stored in the ion gradient. Then, just as water pumped uphill can do the work as it flows back down, (think water wheel or turbine), a substance pumped across the membrane can do work as it leaks back, propelled downhill along the concentration gradient.
What is a sequence of rapidly occurring events that decrease and reverse the membrane potential and then eventually restore it to the resting state?
Action potential or impulses
The diffusion of K out of the cell makes the inside of the cell less positive or more negative and acts to restore the original resting membrane potential This process is called?
repolarization
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What are the steps in the generation of an action potential?
1. electrical signals are sent through nerves. 2. Travels down axon. 3. k+ +Na+ ions flow down concentration gradients to restore equilibrium.
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Describe the events occurring in the cell membrane that permit the conduction of an impulse?
When a nerve impulse is conducted, the neuronal cell membrane undergoes changes in electrical potential. This starts with a rapid influx of sodium ions into the cell through voltage-gated sodium channels, depolarizing the membrane. This depolarization triggers the opening of adjacent sodium channels, resulting in an action potential that travels along the membrane. After the impulse passes, the sodium channels close, and potassium channels open, allowing potassium ions to exit the cell and restore the resting potential.
When does a neuron exhibit resting potential?
The human nervous system consists of billions of nerve cells (or neurons)plus supporting (neuroglial) cells. Neurons are able to respond to stimuli (such as touch, sound, light, and so on), conduct impulses, and communicate with each other (and with other types of cells like muscle cells). Neurons can respond to stimuli and conduct impulses because a membrane potential is established across the cell membrane. In other words, there is an unequal distribution of ions (charged atoms) on the two sides of a nerve cell membrane. The membranes of all nerve cells have a potential difference across them, with the cell interior negative with respect to the exterior (a). In neurons, stimuli can alter this potential difference by opening sodium channels in the membrane. For example, neurotransmitters interact specifically with sodium channels (or gates). So sodium ions flow into the cell, reducing the voltage across the membrane. Once the potential difference reaches a threshold voltage, the reduced voltage causes hundreds of sodium gates in that region of the membrane to open briefly. Sodium ions flood into the cell, completely depolarizing the membrane (b). This opens more voltage-gated ion channels in the adjacent membrane, and so a wave of depolarization courses along the cell - the action potential. As the action potential nears its peak, the sodium gates close, and potassium gates open, allowing ions to flow out of the cell to restore the normal potential of the membrane. Membranes are polarized or, in other words, exhibit a RESTING MEMBRANE POTENTIAL. This means that there is an unequal distribution of ions (atoms with a positive or negative charge) on the two sides of the nerve cell membrane. This POTENTIAL generally measures about 70 millivolts (with the INSIDE of the membrane negative with respect to the outside). So, the RESTING MEMBRANE POTENTIAL is expressed as -70 mV, and the minus means that the inside is negative relative to (or compared to) the outside. It is called a RESTING potential because it occurs when a membrane is not being stimulated or conducting impulses (in other words, it's resting). Source : Internet.
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How is resting potential achieved?
The resting membrane potential difference between the inside and the outside of the cell is the result of selective permeability of the cell membrane and the active transport of ions into and out of the cell. Almost all cells have a potential difference, but some cells, neuron and heart muscle, also have voltage and chemically gated channels that allow for transient deviations from the resting potential.
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