Cell division is regulated by the p53 gene and relates to cancer because Cancer is caused by uncontrolled cell divison
P53 is a type of gene that regulates cell death or apoptosis. It normally inhibits proliferation in cell division, but when it is deactivated by mutations, it can lead to cancer.
The p53 is the tumor suppressor protein that regulates cell cycles and hence has a role in preventing cancer.
If the cell cannot stop dividing then a tumour will form. The tumour can be cancerous or benign. Uncontrolled cell division may be caused by substances called carcinogens (e.g. in cigarettes) which can switch the P53 gene off. This gene is responsible for controlling cell division.
No, when it detects a mutation or damage to the DNA of a cell it codes for a protein that causes apoptosis to occur which is the controlled death of a cell. so it prevents uncontrolled cell division.
Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
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Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
Loss of cell cycle control is typically an oncogenic process. For cancer to occur, the cells need to replicate beyond any normal physiological control. To answer your question, one factor that in important in controlling the cell cycle is p53. p53 is a checkpoint control in the G1 phase of the cell cycle. Defects in p53 cause a loss of cell cycle regulation and are considered an oncogenic transformation.
The p53 Tumour suppressor gene is located on chromosome 17. As its name suggests, it has an anti-tumour activity. That involves the overseeing of the cell cycle. If either the cell or the DNA is impaired, p53 activates the production of specific chemicals (like p21 protein), which interferes with the cell cycle. Depending on the nature/extent of the damage, its either repaired, thus cell cycle is allowed to continue, or p53 initiate apoptosis (cell death). This mechanism would effectively stop the proliferation of infected (malignant) cells, however, p53 itself (or any of genes/chemicals p53 regulates) could be damaged/mutated. In the inherited condition, Li-Fraumeni syndrome, for example, only one functional copy of p53 gene is inherited (instead of two), which interferes with its normal function.