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Whether a hyperthyroid condition may be "cured" depends entirely upon the reason the thyroid is dysfunctional. If excess iodine is the cause, then reducing the amount of iodine in the diet is usually sufficient. If autoimmune thyroid disease is the problem, there is no cure, however there are treatments. If a *hot* thyroid nodule is creating too much thyroid hormone, removing the nodule is often sufficient to elimiate the problem.

AnswerTechnically, there is no cure, because hyperthyroidism is just a symptom of other problems. (There is surgical removal, but this is not as harmless as many would have you believe.) I have provided a link to help you begin your research for alternative treatments in the 'Larry On Hyperthyroidism' link in the related links below. Make certain you avoid iodine, and that means iodized table salt in particular. I cannot believe I am saying this... but the fluoride in tap water can actually help you to suppress the thyroid by the fluoride's neutralization of your body's iodine. I recommend consuming a couple of teaspoons of coconut oil (coconut butter) a day. This helps shield the thyroid from the toxic effects of the bad fats you eat. There almost certainly needs to be a few changes in diet. Avoid all artificial sweeteners (in particular Aspartame) because they will increase your risk of your already increased risk of eventually developing Parkinson's Disease.
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Q: Is there a cure for toxic goiter which is also known as hyperthyroidism?
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An enlargement of the thyroid gland is called a?

A goiter is inflammation of the thyroid gland, also known as the thyroid gland becoming enlarged. Worldwide, the most common cause of a goiter is due to a lack of iodine in our daily diet. In the United States, a goiter more often occurs due to the over or underproduction of thyroid hormones, or due to nodules that develop in the thyroid gland.


What are the symptoms of goiter?

Visible swelling at the base of your neck/throat due to swelling of the thyroid gland. Coughing, trouble breathing, and swallowing are also very common. Since hyperthyroidism is usually associated with goiter, symptoms such as palpitations, increased nervousness, insomnia, muscle weakness, and fatigue can also be present.


The patient also has an enlarged thyroid gland called a?

An enlarged thyroid gland is clinically referred to as goiter and / or bronchocele.This condition should not be confused with hyperthyroidism, which is increased secretion of thyroid hormone.


What is a autoimmune disorder caused by hyperthyroidism?

Grave's Disease and Hashimoto's thyroiditis are two that I know of. Grave's tends to have a steady stream of hyperthyroid symptoms while in Hashimoto's tyroiditis it might start out hyperthyroid and as the antibodies attack it will eventually dwindle down to hypothyroidism. Somtimes there will be bursts of hyperthyroid behavior as the thryoid gland is destroyed by antibodies in Hashimoto's thryoiditis.


What is the pathophysiology of goiter?

PathophysiologyThe histopathology varies with etiology and age of the goiter. Initially, uniform follicular epithelial hyperplasia (diffuse goiter) is present with an increase in thyroid mass. As the disorder persists, the thyroid architecture loses uniformity, with the development of areas of involution and fibrosis interspersed with areas of focal hyperplasia. This process results in multiple nodules (multinodular goiter). On nuclear scintigraphy, some nodules are hot, with high isotope uptake (autonomous) or cold, with low isotope uptake, compared with the normal thyroid tissue. The development of nodules correlates with the development of functional autonomy and reduction in thyroid-stimulating hormone (TSH) levels. Clinically, the natural history of a nontoxic goiter is growth, nodule production, and functional autonomy resulting in thyrotoxicosis in a minority of patients.Signs / symptomsdyspneastridorHemorrhagebronchitisdysphagia.hoarseness.facial plethoraand dilated neck veins


Is goiter genetic?

A goiter is inflammation of the thyroid gland, also known as the thyroid gland becoming enlarged. A goiter isn't necessarily genetic. However, thyroid conditions do run in families. So if someone in your family has a thyroid condition, this can increase your chances of having a thyroid condition.


What condition or disease is characterized by exophthalmos?

The name of the disease is 'Graves' disease'. It is also called as primary hyperthyroidism. You have exophthaloms , pretibial non pitting type of oedema and hyperthyroidism in Graves' disease.


What is the difference between a goiter and nodule of the thyroid?

A goiter is inflammation of the thyroid gland, also known as the thyroid gland becoming enlarged. Meanwhile, a thyroid nodule is a lump in or on the thyroid gland.


Which condition is also known as thyromegaly?

goiter also means enlargement of the thyroid gland.


What causes a colloidal or endemic goiter?

Colloid nodular goiters are also known as endemic goiters. They are usually caused by not getting enough iodine in the diet.


What is hypothyroidism and hyperthyroidism?

When the thyroid doesn't produce enough thyroid stimulating hormones it becomes underactive, which is also known as hypothyroidism. When the thyroid produces too much thyroid hormone it becomes overactive, which is also known as hyperthyriodism.


Toxic nodular goiter?

DefinitionToxic nodular goiter involves an enlarged thyroid gland that contains a small rounded growth or growths called nodules. These nodules produce too much thyroid hormone.Alternative NamesToxic multinodular goiter; Plummer's diseaseCauses, incidence, and risk factorsToxic nodular goiter arises from an existing simple goiter. It occurs most often in the elderly. Risk factors include being female and over 60 years old. This disorder is never seen in children.Sometimes patients with toxic multinodular goiter will develop high thyroid levels for the first time after they receive a large amount of iodine through a vein (intravenously). The iodine may be contrast for a CT scan or heart catheterization.SymptomsSymptoms are those of hyperthyroidism. However, the bulging eyeballs seen in Graves disease do not occur.Symptoms include:FatigueFrequent bowel movementsHeat intoleranceIncreased appetiteIncreased sweatingMenstrual irregularities (in women)Muscle crampsNervousnessRestlessnessWeight lossSigns and testsA physical examination will show one or many nodules in the thyroid. There may be a rapid heart rate.Other test results:Serum thyroid hormone levels (T3, T4) are high.Serum TSH(thyroid stimulating hormone) is low.Thyroid scanshows that the nodules are taking up increased amounts of radioactive iodine.TreatmentTreatments include:Antithyroid drugs (propylthiouracil, methimazole)Radioactive iodineSurgeryBeta blockers (propranolol) can control some of the symptoms of hyperthyroidism until thyroid hormone levels in the body are under control.Expectations (prognosis)Because toxic nodular goiter is mainly a disease of the elderly, other chronichealth problems may influence the outcome of this condition. The elderly person may be less able to tolerate the effect of hyperthyroidism on the heart.ComplicationsHeart complications:Atrial fibrillationCongestive heart failureRapid heart rateOther complications:Bone loss leading to osteoporosisThyroid crisis or storm is an acute worsening of hyperthyroidism symptoms. It may occur with infection or stress. Thyroid crisis may cause:Abdominal painDecreased mental alertnessFeverImmediate hospitalization is necessary.Complications of having a very large goiter may include difficulty breathing. This is due to pressure on the airway passage that lies behind the thyroid.Calling your health care providerCall your health care provider if you have symptoms of this disorder. Follow the health care provider's recommendations for follow-up visits.PreventionTo prevent toxic nodular goiter, treat hyperthyroidism and simple goiter as your health care provider recommends.ReferencesLadenson P, Kim M. Thyroid. In: Goldman L and Ausiello D, eds. Cecil Medicine. 23rd ed. Philadelphia, Pa: Saunders; 2007:chap 244.Davies TF, Larsen PR. Thyrotoxicosis. In: Kronenberg HM, Melmed S, Polonsky KS, Larsen PR, eds. Williams Textbook of Endocrinology. 11th ed. Philadelphia, Pa: Saunders Elsevier;2008:chap 11.