Want this question answered?
False. Coagulation starts almost instantly after an injury and ends in a fibrin clot.
Platelet
thrombopoietin
a blockage
calcium is the impotant ion in coagulation The steps to coagulation: 1) Vasospasm: contraction of smooth muscle in the vessel wall, the severed ends may close completely. The body's reaction to the loss of blood is to seal off the hemoraging part completely. An example of this is the movie "Black Hawk Down" after the guy gets shot in the leg, and the medic says "I cant find the femoral artery" 2) Platelet Plug Formation: Platelets start to stack on top of each other to form rough surfaces to start clotting. 3) Coagulation 4) Fibrin strands form to collect blood cells and more platlets to stimulate clotting 5) Clot formation
Vasoconstriction, platelet plug formation and clotting of blood.
False. Coagulation starts almost instantly after an injury and ends in a fibrin clot.
haemorrhage prevented by platelet aggregation and agglutination (clotting) and vasoconstriction to maintain blood pressure/perfusion pressure. formation of clot through platelet clumping and binding with protein fibrin.
Platelet
the three steps of hemostasis are :- (1) vasoconstrictiion (2) blood platelet plug formation (3) coagulation of blood
vascular, platelet, coagulation, clot retraction, clot destruction
thrombopoietin
a blockage
Hemostasis is when the blood is outside the body by way of an injury. The vessels constrict and cause clotting to stop the bleeding.
Primary hemostasis is the overall sequence of events that leads to the formation of a platelet plug, while Secondary hemostasis is the signal transduction pathway that leads to the formation of fibrin. Secondary Hemostasis is always a part of Primary hemostasis, but involves many clotting factors and other signaling molecules that are specific to the formation of fibrin
Because coagulation requires a combination of platelet activation, activation of coagulation cascade (by trauma or damaged endothelial walls), and in predisposed individuals, abnormalities in regulator proteins such as protein C and anti-thrombin
1. vascular spasm 2. platelet phase 3. coagulation 4. retraction 5. fibrinolysis