it is the amount of blood available for the heart to pump when the ventricles contract
cardiac output
A change in cardiac output without any change in the heart rate, pulmonary artery wedge pressure (PAWP = equated to preload) or systemic vascular resistance (SVR = afterload) would have to be due to a change in the contractility of the heart. Cardiac output (CO) is roughly equal to stroke volume x heart rate. Stroke volume is related to preload, contractility, and afterload. As you can see, the only variables you have not controlled for is cardiac contractility.
Cardiac contractility is the force of contraction possible for any given length of the cardiac muscle. It is related to the intracellular calcium levels.
John is about to preload his truck with logs.
it decreases blood volume and preload
Preload is caused by decreased blood volume in ventricles. Therefore, decreased preload directly caused by bleeding, polyuria, dehydration.
if you find out tell me what preload is
Preload is the amount the pinion bearings are squeezed together by the pinion nut.
Severe dehydration will decrease preload because there won't be as much volume coming into the heart.
It decreases preload and afterload as a result of the dilation in the venous and arterial vasculature from the nitric oxide.
degree of ventricular myocardial stretch; end diastolic volume. the heart cannot send no more blood. doctors put the patient on medication. there is a lot of pressure. the ventricle already pumps the blood.
Cardiac output (CO) is determined by the heart rate (HR) and the volume of blood pumped by each beat (stroke volume - SV). Mathematically, cardiac output can be represented by the equation: CO = HR x SV As such, if total cardiac output falls as a result of decreased stroke volume, the heart rate can increase to keep the total cardiac output normal, to a certain extent. Stroke volume is more complicated; it is determined by many different factors, including preload, afterload, competence of the atrioventricular valves, ventricular cavity size, and the strength of the squeeze of the cardiac muscle, amongst others. Any change in one of these factors requires a compensation in one or more of the others to maintain cardiac output.