Ether prevents the action potential, by opening potassium ion pores, which allows the escape of potassium from the neurons, which results in hyper-polarization of the neuron, thus preventing the action potential from occurring.
Lidocaine is an antagonist inhibitor that blocks sodium ion channels, when these are blocked there's no action potential (nerve impulses).
Ether blocked the impulse transmission.
It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,and it can mask digitalis toxicity.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
A synaptic potential exists at the INPUT of a neuron (dendrite), and an action potential occurs at the OUTPUT of a neuron (axon). (from OldGuy)(from Ilantoren:) A synaptic potential is the result of many excitatory post synaptic potentials (epsp) each one caused by the synaptic vesicles released by the pre-synaptic terminus. If there are enough of these epsp then the responses will summate and depolarize the post-synaptic membrane at the axon hillock enough to fire an action potential.
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It creates an action potential
They both decrease action potential duration, but TTX is the only one that decreases the maximum rate of depolarization.
Depends on disease
Ether blocked the impulse transmission.
what effect does the drug quabain have on neuron
It can prolong the cardiac action potential. It can also have other effects, such as torsades de pointes,and it can mask digitalis toxicity.
No, they are two different substances. Lidocaine was given the -caine ending only due to the anesthetic effect it shares with cocaine. However, sometimes cocaine is adulterated with lidocaine to increase the perceived potency of the product (due to lidocaine's numbing effects).
A synapse and an action potential have a flip-flopping cause and effect relationship, in that an action potential in a presynaptic neuron initiates a release of neurotransmitters across a synapse, which can then subsequently potentially trigger an action potential in the axon of the postsynaptic neuron, which would then cause release of neurotransmitters across a following synapse.
Ether causes potassium ion pores to open, allowing potassium ions to leave the neuron, hyper-polarizing the neuron so it is unable to fire an action potential.
It blocks the sodium channels that are required to create action potential in the muscles to make them contract.
It makes the muscle totally relax as it blocks the action potential in the nerves.
A negative effect is any result of an action which is perceived to be detrimental. This means its a subjective opinion. What might be negative to you may be positive to someone else.