opening of slow calcium channels
An action potential is not passively propagated down the axon. There have to be ion channels along the axon or else the action potential will gradually decay. So the the rate of that the action potential 'travels' is dependent on the passive property called the length constant of the axon (factor in capacitance, axon diameter) plus the density of ion channels.
In the fast response action potential of cardiac muscle, the sarcolemma rapidly depolarizes and reaches a plateau. In the plateau phase, Ca2+ released by the glycocalyx comes into the muscle cell from the extracellular fluid. This Ca2+ is called "trigger Ca2+" because it induces the release of Ca2+ from the sarcoplasmic reticulum.
Tetrodotoxin blocks action potentials by binding with the voltage-gated, fast sodium (Na+) channels in neural membranes. This prevents the influx of sodium ions required to propagate an action potential.
To speed up transmission of the action potential from where it originates (axon hillock) to where it ends (axon terminal), the action potential propagates by 'saltatory conduction' - and the structure that makes this possible is the insulating layer of myelin sheath that wraps around the axon, arranged in 'nodes' along its length. Technically, it's the gaps between the nodes (nodes of Ranvier) that cause the action to continually propagate and maintain its fast conduction velocity.
The velocity of propagation of an action potential depends on axoplasm resistance and membrane resistance. Axoplasm resistance explains how fast a charge can move within an axon. The larger the diameter of the axon, the more quickly it can pass through. Membrane resistance describes how permeable the membrane is to the ion. The less permeable, the faster the propagation of the action potential. Therefore, myelination increases the membrane resistance and ultimately allows for fast propagation. In demyelinating diseases, there is little or sometimes no myelin covering the axons. In these cases action potentials will slow down or completely cease.
increased membrane permeability to sodium ions
An action potential is not passively propagated down the axon. There have to be ion channels along the axon or else the action potential will gradually decay. So the the rate of that the action potential 'travels' is dependent on the passive property called the length constant of the axon (factor in capacitance, axon diameter) plus the density of ion channels.
The rising action in "Fast Sam, Cool Clyde, and Stuff" involves the planning and execution of Sam and Clyde's bus trip from Harlem to the 1964 New York World's Fair. As they encounter obstacles and make decisions along the way, the tension and conflict build leading up to the climax of the story - when they finally reach the fair.
In the fast response action potential of cardiac muscle, the sarcolemma rapidly depolarizes and reaches a plateau. In the plateau phase, Ca2+ released by the glycocalyx comes into the muscle cell from the extracellular fluid. This Ca2+ is called "trigger Ca2+" because it induces the release of Ca2+ from the sarcoplasmic reticulum.
There is no way of running on Dead Rising 2
no, fast is an adjective.
Tetrodotoxin blocks action potentials by binding with the voltage-gated, fast sodium (Na+) channels in neural membranes. This prevents the influx of sodium ions required to propagate an action potential.
To speed up transmission of the action potential from where it originates (axon hillock) to where it ends (axon terminal), the action potential propagates by 'saltatory conduction' - and the structure that makes this possible is the insulating layer of myelin sheath that wraps around the axon, arranged in 'nodes' along its length. Technically, it's the gaps between the nodes (nodes of Ranvier) that cause the action to continually propagate and maintain its fast conduction velocity.
The velocity of propagation of an action potential depends on axoplasm resistance and membrane resistance. Axoplasm resistance explains how fast a charge can move within an axon. The larger the diameter of the axon, the more quickly it can pass through. Membrane resistance describes how permeable the membrane is to the ion. The less permeable, the faster the propagation of the action potential. Therefore, myelination increases the membrane resistance and ultimately allows for fast propagation. In demyelinating diseases, there is little or sometimes no myelin covering the axons. In these cases action potentials will slow down or completely cease.
not very fast actually, it is a bolt action sniper rifle and if you want to fire it fast you better be able to reload a bolt action fast
chloroform
The Kodak DCS 720x Pro SLR is great for fast action shots.