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In the fast response action potential of cardiac muscle, the sarcolemma rapidly depolarizes and reaches a plateau. In the plateau phase, Ca2+ released by the glycocalyx comes into the muscle cell from the extracellular fluid. This Ca2+ is called "trigger Ca2+" because it induces the release of Ca2+ from the sarcoplasmic reticulum.
No, calcium itself is not a neurotransmitter BUT it is highly important in the process of the action potential. The action potential triggers the influx of calcium at the end of the terminal bouton, causing the influx of Ca2+ into the cell and this triggers for the release of the neurotransmitter. :)
Ca2 is basically two Ca's so it's larger (the plus doesn't compensate)
Once you have the action potential made from the influx of Na traveling down the axon depolarizing it. The action potential reaches the axon terminals, the depolarization causes Ca2+ to enter the cell and that causes the release of the neurotransmitters out of the axon terminals and into the dendrites of the next axon to continue the signaling pathway.
intracellular levels of Ca2+
The voltage-gated Ca2+ channels are opened when an action potential releases neurotransmitters from a neuron. A neuron transmits nerve impulses.
In the fast response action potential of cardiac muscle, the sarcolemma rapidly depolarizes and reaches a plateau. In the plateau phase, Ca2+ released by the glycocalyx comes into the muscle cell from the extracellular fluid. This Ca2+ is called "trigger Ca2+" because it induces the release of Ca2+ from the sarcoplasmic reticulum.
No, calcium itself is not a neurotransmitter BUT it is highly important in the process of the action potential. The action potential triggers the influx of calcium at the end of the terminal bouton, causing the influx of Ca2+ into the cell and this triggers for the release of the neurotransmitter. :)
Slow inward diffusion of Ca2+
Ca2 is basically two Ca's so it's larger (the plus doesn't compensate)
Gap Junctions in the cell spread the action potential to nearby cells.
Once you have the action potential made from the influx of Na traveling down the axon depolarizing it. The action potential reaches the axon terminals, the depolarization causes Ca2+ to enter the cell and that causes the release of the neurotransmitters out of the axon terminals and into the dendrites of the next axon to continue the signaling pathway.
Ca2+ and Br- obviously must form, CaBr2 Calcium bromide
An isotope of calcium
intracellular levels of Ca2+
they cannot bond together, they are separate .
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