Excitation begins as a motor neuron transmits an action potential to the neuromuscular junction where it gets propagated along the muscle cell. This action potential is an electrical impulse that depolarizes the muscle cell membrane, the sarcolemma, which then releases ions in the sacroplasmic reticulum and transverse tubules. When this happens calcium is released into the sarcomeres that contain actin and myosin myofilaments. The calcium unlocks the binding sites and creates crossbridges with the globular heads on the mysoin fibers. With ATP as the energy source, this crossbridging brings the Z-lines closed together and the muscle fiber contracts.
Axon of neuron, sarcolemma, and T-tubules
Myofibrils
controlled output of action potentials, progressive recruitment of motor units, control of the rate of attachment/reattachment of actin and myosin heads and the power stroke rate
Skeletal or voluntary muscle is capable of rapid contraction and is responsible for skeletal movement.
yes skeletal muscle stretch by means of contraction.
Low calcium levels in the extracellular fluid increase the permeability of neuronal membranes to sodium ions, causing a progressive depolarization, which increases the possibility of action potentials. These action potentials may be spontaneously generated, causing contraction of skeletal muscles (tetany).
C. neuromuscular junctions
stimulation of the muscle by a nerve ending.
during skeletal muscle contraction ,I band and H zone shortens. Sarcomeres
The skeletal muscles do have the involuntary muscle because they help it in the contraction process.
neuromuscular junctions Action potentials conduct down T tubules into skeletal muscles
smooth skeletal cardiac and contraction mbroro
The Sarcomere