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Q: What laboratory values would indicate a patient has experienced an acute myocardial infarction?
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An enlarged R wave on an ECG would indicate?

< A myocardial infarction.


What do cyanosis and increased breathing rate indicate?

It could be either a Myocardial Infarction (heart attack) or a form of shock.


How should a patient prepare for a creatine kinase test?

Elevated levels indicate damage to either muscle or brain; possibly from a myocardial infarction (heart attack), muscle disease, or stroke.


Why Blood sugar testing are more important than Blood Pressure?

Because early intensive glucose lowering not only indicate the end of diabetes but also reduction in mortality from chances of myocardial infarction.


What is ST abnormality?

ST abnormality refers to abnormal changes in the ST segment of an electrocardiogram (ECG). It can indicate myocardial ischemia, injury, or infarction. ST abnormalities are important indicators of heart conditions and often prompt further evaluation and treatment.


What is the purpose of a creatine kinase test?

Elevated levels indicate damage to either muscle or brain; possibly from a myocardial infarction (heart attack), muscle disease, or stroke.


What is the diagnostic procedure for myocardiac infarction?

The myocardium is the muscle of the heart, so myocardial inflammation would be swelling of this tissue, for instance. A myocardial infarction is also known as a heart attack, where blood flow is blocked to the myocardium. The type of heart attack would depend on what abnormalities were found on EKG (ie: ST elevation=ST Elevation Myocardial Infarction, or STEMI).


Cheek numbnessbut also chest discomfort and sharp pain to the shoulder blades?

Cheek numbness associated with chest discomfort and sharp pain to the shoulder blades could indicate a heart attack or myocardial infarction. These symptoms might also indicate a pinched nerve or pulled muscle or even spinal injury. It is best to check with a physician regarding symptoms of chest discomfort.


Why does a prominent Q wave in an ECG indicate old infarction?

because it does, so just accept it


What takes place as a result of a myocardial infarction?

Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated myocardial infarction and ST-elevated myocardial infarction, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Blood stream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote a thrombus (blood clot) that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary vasculature, it leads to myocardial infarction (necrosis of downstream myocardium). If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells die (chiefly through necrosis) and do not grow back. A collagen scar forms in its place. Recent studies indicate that another form of cell death called apoptosis also plays a role in the process of tissue damage subsequent to myocardial infarction.[33] As a result, the patient's heart will be permanently damaged. This scar tissue also puts the patient at risk for potentially life threatening arrhythmias, and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life threatening arrhythmia is ventricular tachycardia (V-Tach/VT), which may or may not cause sudden cardiac death. However, ventricular tachycardia usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. The cardiac defibrillator is a device that was specifically designed to terminate these potentially fatal arrhythmias. The device works by delivering an electrical shock to the patient in order to depolarize a critical mass of the heart muscle, in effect "rebooting" the heart. This therapy is time dependent, and the odds of successful defibrillation decline rapidly after the onset of cardiopulmonary arrest.


What aftercare is required after the creatine kinase test?

There is typically no specific aftercare required after a creatine kinase (CK) test. The test itself is a simple blood draw, and most people can resume their usual activities immediately after. It is always advisable to follow any instructions given by the healthcare provider, such as avoiding strenuous exercise if recommended.


What does an elevated troponin T level mean?

Cardiac troponin levels are usually too low to measure. Elevated troponin T levels indicate that there has been some form of damage to the heart.Strenuous exercise can also temporarily elevate troponin levels for a brief time. The effects of a heart attack would keep troponin T levels elevated for up to two weeks.