Ask anyone with insulin-dependent Diabetes. Some or all of the following would happen:
* Glucose from the blood would be unable to enter the body's tissues, since insulin is required each time that happens.
* Cells in the body would be denied glucose, their primary fuel. Some cells would die, others like brain cells would move to a secondary fuel (ketone bodies).
* Glucose remaining in the blood would accumulate, rising higher and higher.
The sugar levels in blood will rise up really fast.
If the insulin receptors stopped working the blood sugar will raise which may leads to diabetes
The liver and kidneys destroy insulin after it has its effect. The pancreas is the site of insulin production.
Your pancreas knows how much insulin it needs to secrete if you take additional insulin insulin receptors in fat cells will become less responsive to it, so your pancreas will need to secrete more for the same effect. This process is the begining of type 2 diabetes. You would need to take additional insulin for a few weeks without any break for this to occure.
insulin
Insulin allows glucose(sugar) to enter the body's cells. Insulin also stimulates the liver to convert glucose to glycogen and store it for later use. The end effect is blood sugar is decreased.
It doesn't atropine only acts on muscarinic receptors (it's a competitive antagonist here for ACh), but histamine acts on different receptors (histamine receptors). There is no direct interaction between atropine and histamine receptors
decrease blood sugar
Yes, weight gain is a reasonably common side effect of insulin therapy.
Insulin handles the absorption of sugar. Carbohydrates are sugars.
If you put insulin into food it would not have the same effect. You see, insulin needs to be in the blood stream. That is why you have to inject it with a syringe.
Weight Gain Is a common side effect of Insulin
the receptors on the postsynaptic membrane