high fowler's position
decrease edema
No it is not. It is considered a beta blocker which works to decrease blood pressure and decrease heart rate.
it decreases blood volume and preload
No, Thiazide Diuretics actually increase renal excretion of Phosphate. They inhibit the sodium-chloride symporter in the distal convoluted tubule of the kidney, leading to increased excretion of sodium, chloride, and also phosphate.
They are not inherently either. They can be good if they are used properly. For example, because diuretics increase urine output and decrease blood volume, if someone with high blood pressure takes some prescribed diuretics they can decrease their blood volume and thus their blood pressure. This can be very beneficial. However, if someone already has a blood pressure that is too low, possibly after they have been bleeding out, then taking diuretics would further decrease their BP and this could result in insufficient blood flow to the brain resulting in syncope, brain damage, or death
Patients with pulmonary edema may undergo phlebotomy procedures to decrease their total blood volume.
Chronic pulmonary diseases (most likely restrictive, but could also be constrictive), inadequate surefactant production, pulmonary edema, thoracic trauma (think fractured ribs). The list goes on.
Drugs that decrease PTH include cimetidine and propranolol.
Severe aortic insufficiency can be treated with medical therapy. Pharmaceuticals to decrease blood pressure, with diuretics and vasodilators, are helpful in patients with aortic insufficiency.
Vitamins E and B6 may decrease breast tenderness and help with fatigue and mood swings. Diuretics that remove excess fluid from the body and pain relievers may also help.
some diuretics (like lasix, hydrochlorothiazide) lead to an increased loss of potassium with the urine. In some people this leads to hypokalemia after a usually prolonged interval (months). There are other diuretics and hypertension medications which decrease this effect and with a combination some people can regain a normal potassium balance. (kalium = potassium). Others may have to stop taking it, or, alternatively, take extra potassium.
The connection between diuretics and hyperglycemia involves intracellular K+ levels. Intracellular K+ is involved in the secretion of a lot of hormones...including insulin. Some diuretics cause hypokalemia, (decrease in K+). This inhibits insulin secretion and can lead to hyperglycemia. It's not really a problem unless the patient is pre-diabetic. In that case, it is significant enough to push them over to diabetes. Hope this helped!