No, this is not true. FSH starts to suppress during pregnancy. LH remains low throughout lactation, but begins to rise again during weaning.
Osteoclasts activity is inhibited and blood calcium levels rise.
High FSH levels are normal in a post menopausal women and can't be used to diagnose hair loss.
LH and FSH levels are genetically predetermined.
Water levels rise and damage can come to buildings.
Anorexia nervosa and disorders of the hypothalamus or pituitary gland can result in abnormally low FSH levels. Abnormal levels can also indicate: infertility hypopituitarism klinefelter syndrome (in men) turner syndrome ovarian failure
Follicle Stimulating Hormone stimulates follicle and egg maturation in females and stimulates sperm production and maturation in males. Therefore low levels of FSH slows spermatogenesis.
In the beginning of the menstrual cycle, LH and FSH stimulate the ovaries to make estrogen. During this time, there is a negative feedback loop, so levels of all three hormones are rather low. Eventually, as the estrogen levels slowly creep up, there comes a time when it switches from negative feedback to positive feedback (not exactly sure what the biochemical basis of this is), and LH and FSH levels skyrocket (estrogen levels go up to, but not as sharply, and there is always more LH than FSH). The LH surge causes ovulaton. The corpus luteum starts secreting progesterone, which inhibits LH and FSH secretion in a negative feedback manner, and so FSH and LH levels drop sharply.
When a person suffers an injury, prolactin and dopamine levels increase. Meanwhile, insulin, thyroid hormones, FSH, LH, estrogen, and testosterone decrease.
it reduces the levels of FSH - negative feedback loop
FSH is the major survival factor that rescues the follicles from atresia (programmed death of the somatic cells of the follicle and oocyte). In the luteal-follicle phase transition period the serum levels of progesterone and estrogen (primarily estradiol) decrease and no longer suppress the release of FSH, consequently FSH peaks at about day three (day one is the first day of menstrual flow). The cohort of small antral follicles is normally sufficiently in number to produce enough Inhibin B to lower FSH serum levels. As a woman nears perimenopause the number of small antral follicles recruited in each cycle diminishes and consequently insufficient Inhibin B is produced to fully lower FSH and the serum level of FSH begins to rise. When the follicle matures and reaches about 10 mm in diameter it starts to secrete significant amounts of estradiol. Normally in humans only one follicle becomes dominant and survives to ovulate, the remaining follicles in the cohort undergo atresia. The increase in serum estradiol levels cause a decrease in FSH production by inhibiting GnRH production in the hypothalamus. The decrease in serum FSH level causes the smaller follicles in the current cohort to undergo atresia as they lack sufficient sensitivity to FSH to survive. Occasionally two follicles reach the 10 mm stage at the same time by chance and as both are equally sensitive to FSH both survive and grow in the low FSH environment and thus two ovulations can occur in one cycle possibly leading to non identical twins.
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