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Affinity for different agonists, their locations throughout the body, the effects produced from their activation and supression. Presuming your talking about adrenergic receptorsAlpha adrenergic receptors & beta adrenergic receptors produce vasoconstriction and vasodilation respectively.
Cholinergic
Beta-adrenergic blocking agents include propranolol (Inderal), atenolol (Tenormin), and pindolol (Visken).
There are at lest 3 types of beta receptors and they are found in different organs. Beta-1 (β1) receptors are found in the heart, eye, and kidneys while beta (β2) receptors are found in the lungs, gastrointestinal tract, liver, uterus, blood vessels, and skeletal muscle. The third type, beta (β3) receptors are found in fat cells.
To understand how beta adrenergic blockade lowers blood pressure, you need to understand a little physiology. Beta receptors are present in many different places in the body: the heart, the blood vessels, the kidneys, the lungs, the muscles, etc. They perform different tasks in each different place as well, and there are different classes of receptors. For instance, in the heart, the receptors are called beta-1 receptors and they increase the rate of firing of the SA node (chronotropy), increases cardiac conduction velocity (dromotropy) and increase the strength with which the heart beats (inotropy). In the arterioles, the receptors cause dilatation of the vessels and are called beta-2 receptors. In the kidneys, beta-1 receptors increase renin output from the juxtamedullary cells when stimulated. Renin is an enzyme that produces a potent vasocontrictor when it activates the angiotensin cascade. Now, how does beta antagonism lower blood pressure. First, in the heart, beta blockade causes a slowing of the heart rate and stroke volume, effectively decreasing cardiac output. In the kidneys, beta blockade decreases renin release, which decreases the amount of systemic vasoconstrictors in the body. These two effects cause the blood pressure to decrease over time.
beta receptors
Affinity for different agonists, their locations throughout the body, the effects produced from their activation and supression. Presuming your talking about adrenergic receptorsAlpha adrenergic receptors & beta adrenergic receptors produce vasoconstriction and vasodilation respectively.
Cholinergic
Beta adrenergic agents serve as inhibitors that prevent beta adrenergic substances (neurotransmitters) such as epinephrine from binding to beta adrenergic receptors (beta 1, beta 2 and beta 3). This prevents the effects of the neurotransmitters thereby reducing blood pressure and heart rate.
alpha 1, alpha 2, beta 1, beta 2
The alpha adrenergic receptors are located in the effector organs of sympathetic nervous system. They are the most common type of alpha adrenergic receptor. Alpha adrenergic receptors tend to be excitatory. They have a greater affinity for nonepinephrine than epinephrine.
The neurotransmitter for sympathetic nervous system is norepinephrine (or also called noradrenaline) that acts on adrenergic receptors of the effector organ (alpha 1, beta 1, beta 2 receptors). The adrenergic receptors are G-protein coupled.
Propranolol acts on the beta-adrenergic receptors anywhere in the body, and has been used as a treatment for emotional anxiety and rapid heart beat.
Beta 2 adrenergic receptors in lung tissue cause dilation of bronchioles via sympathetic stimulation.
It is a beta-adrenergic blocking drug
Beta-adrenergic blocking agents include propranolol (Inderal), atenolol (Tenormin), and pindolol (Visken).
Beta blockers, also known as beta-adrenergic blockers