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Why Euthyroid patients with goitres are treated with thyroxine?
Wiki User
∙ 14y ago
It depends on the specific type of goiter. Thyroid physiology and pathophysiology is very complex but I will try to be very basic. Iodine deficiency & low production of thyroxine are the main causes for goitres in euthyroid individuals.
In mountainous areas dietary iodine deficiency still exists, in some areas as 'endemic goitre' where goitre, occasionally massive, is common. The patients may be euthyroid or hypothyroid depending on the severity of iodine deficiency. Because iodine is essential for the thyroid hormone production.
The mechanism is thought to be borderline hypothyroidism leading to TSH stimulation and thyroid enlargement in the face of continuing iodine deficiency.
TSH (Thyroid Stimulating Hormone) is secreted from the anterior pituitary gland and travels to the thyroid gland, "stimulating" the gland. Among the multiple effects it has on the gland, TSH causes cells of the gland to increase size and number. This can contribute to a goiter.
Thyroxine is a thyroid hormone. It is secreted from the thyroid gland and travels throughout the body affecting many organs. It also travels back to the pituitary gland and causes the pituitary to decrease secretion of TSH since the body has plenty of thyroid hormone flowing. By decreasing TSH, thyroxine also helps to decrease growth of the thyroid gland. Normal Serum thyroxine 51.4-154.4 nmol/L. In certain occasions patients are with low levels of thyroxine ,even though they are within normal range; euthyroids. Therefore TSH secretion is not inhibited, resulting the goitre due to the continuous stimulation of the gland.
Wiki User
∙ 14y ago
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