Blood pressure.
•Increased contraction in response to multiple stimuli of same strength
Pulsus paradoxus occurs due to a significant drop in blood pressure during inhalation, typically seen in conditions like cardiac tamponade, severe asthma, or chronic obstructive pulmonary disease (COPD). During inhalation, increased intrathoracic pressure can impair venous return to the heart, leading to reduced stroke volume and subsequently lower systolic blood pressure. This phenomenon is characterized by a difference of more than 10 mmHg in blood pressure readings between inhalation and exhalation. It reflects underlying pathophysiological changes in the cardiovascular system in response to respiratory mechanics.
Increase in Venous returns increases end diastolic volume (EDV). This increase in volume stretch the atrial and ventricular walls. This also stretches the SA nodal fibers and AV nodal fibers triggering a homeostatic response to increase heart rate by up to 15-30% depending on individual and other factors.
Prostoglandins are chemical mediators. These are released from mammalian cells and effect the cells in their immediate viccinity. They produce what's known as an inflammatory response. Prostoglandins are found in cell membranes, they are released due to injury. They cause the dilation of small arteries and arterioles and increase the permeability of the capillaries. They affect neurotransmitters and blood pressure and hence pain sensation.
Hyperaemiaorhyperemiais the increase of blood flow to different tissues in the body. It can have medical implications, but is also a regulatory response, allowing change in blood supply to different tissues throughvasodilation. Clinically, hyperaemia in tissues manifest aserythema, because of the engorgement of vessels with oxygenated blood.[1]Hyperaemia can also occur due to a fall in atmospheric pressure outside the body.
During exercise, the normal blood pressure response is an increase in both systolic and diastolic blood pressure. This is a natural response to the body's increased demand for oxygen and nutrients during physical activity.
Arterioles do not absorb anything. They are small blood vessels that regulate blood flow and blood pressure by constricting or dilating in response to various signals in the body.
The juxtaglomerular cells located in the walls of the afferent arteriole secrete renin when they sense a drop in blood pressure. Renin is released into the bloodstream in response to decreased blood flow to the kidneys, ultimately leading to the activation of the renin-angiotensin-aldosterone system to help regulate blood pressure and fluid balance.
Many things including the short term/acute effects of exericse and the Long term effects which are;Musculoskeletal response: increased blood supply; increase in muscle pliability; increased range of movement;muscle fibre micro tearsEnergy systems: phosphocreatine; lactic acid; aerobic; energy continuum; energy requirements of differentsport and exercise activitiesCardiovascular response: heart rate anticipatory response; activity response; increased blood pressure;vasoconstriction; vasodilationRespiratory: increase in breathing rate (neural and chemical control); increased tidal volumeCardiovascular system: cardiac hypertrophy; increase in stroke volume; increase in cardiac output, decreasein resting heart rate; capillarisation; increase in blood volume; reduction in resting blood pressure; decreasedrecovery time; increased aerobic fitnessMuscular system: hypertrophy; increase in tendon strength; increase in myoglobin stores; increased numberof mitochondria; increased storage of glycogen and fat; increased muscle strength; increased tolerance tolactic acidSkeletal system: increase in bone calcium stores; increased stretch in ligaments; increased thickness of hyalinecartilage; increased production of synovial fluidRespiratory system: increased vital capacity; increase in minute ventilation; increased strength of respiratorymuscles; increase in oxygen diffusion rateEnergy systems: increased aerobic and anaerobic enzymes; increased use of fats as an energy source(taken from a website - not my own work)
Increased intracranial pressure can lead to changes in blood pressure due to the body's compensatory mechanisms. Typically, there is an initial increase in blood pressure in response to increased intracranial pressure to maintain cerebral perfusion. However, as intracranial pressure continues to rise, blood pressure may decrease due to impaired cerebral autoregulation and compromised blood flow to the brain. Overall, the relationship between intracranial pressure and blood pressure is complex and can vary depending on individual factors and the underlying cause of increased intracranial pressure.
That would be increased respiration and increased oxidation. .
It should increase high blood pressure
myogenic mechanism
The two activities that are not increased during symphathetic response are abstract and methods. These are the too main ones.
Increase in the size of an organ caused by an increase in the size of its cells is referred to as hypertrophy. This process can occur in response to increased workload or hormonal stimulation, leading to enhanced function of the organ. Unlike hyperplasia, which involves an increase in the number of cells, hypertrophy focuses on the enlargement of existing cells. Common examples include muscle hypertrophy from strength training and cardiac hypertrophy from increased blood pressure.
A manometer's sensitivity can be increased by increasing the height differential between the two limbs of the manometer, using a liquid with low density, and increasing the diameter of the manometer tube. These factors can help increase the deflection of the liquid in response to pressure changes, improving sensitivity.
Increased blood pressure. This is due to increased sodium in the blood, increasing the total extracellular fluid volume