Pilocarpine is actually a Muscarinic cholinergic receptor (mAChR) agonist. It works on the Parasypathetic nervous system; therefore, it makes sense that the agonist working on the PNS would result in decreased heart rate. Atropine, in contrast, is an muscarinic cholinergic receptor antagonist and will lead to increased heart rate due to the lowered effect of the parasympathetic system on the heart.
Agonist is muscarine and antagonist is atropine.
agonist : trapizius antagonist: latissimus dorsi
Antagonist activity is the activity that is counter to the agonist or it can be viewed as an inhibitory activity. If pharmacology, the antagonist does not have any activity, but it blocks or inhibits the activity of the agonist.
the wrist flexors and wrist extensors
The SSRI class is neither antagonist nor agonist. They are re-uptake inhibitors. To the extent of my knowledge (I am NOT a doctor), a serotonin: agonist enhances production of sertonin at the source neuron, antagonists blocks binding of serotonin to the target neuron effectively blinding it, re-uptake inhibitors block the destruction of serotonin, effectively letting it act for longer.
No, Pilocarpine is an agonist in how it effects heart rate activity (it increases HR).
Agonist is muscarine and antagonist is atropine.
Agonist
Antagonist: lats Agonist: Abdominals
Antagonist
agonist : trapizius antagonist: latissimus dorsi
The Buprenorphine opioid is classified as a partial agonist antagonist.
anteriordelts andtriceps
pilocarpine as a muscuranic agonist, increases tone and motility of intestinal muscles and of urinary tract and billiary duct's smooth muscles.
agonist- Facilitstes synthesis
Drugs can act as agonist or antagonist to regulate a process or function of cellular/chemical process.
Agonist - Pectoralis Major, Triceps Brachii Antagonist - Bicep Brachii, Latissimus Dorsi