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The tropomyosin molecule blocks the active sites of the actin. Troponin is a molecule that is bound to the tropomyosin. Troponin needs CA+ (calcium ions) to bind to it in order to rotate the tropomyosin molecule and expose the actin molecules for the myosin heads to interact for muscle contraction.

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When the sarcomere is at rest what is covering the active sites on actin?

When the sarcomere is at rest, the active sites on actin are covered by tropomyosin molecules. Tropomyosin blocks the myosin-binding sites on actin, preventing cross-bridge formation and muscle contraction.


How does allosteric regulation differ from noncompetitive inhibition in terms of their mechanisms of action on enzyme activity?

Allosteric regulation involves a molecule binding to a site on the enzyme other than the active site, causing a conformational change that either activates or inhibits the enzyme. Noncompetitive inhibition involves a molecule binding to a site other than the active site, but it does not cause a conformational change. Instead, it blocks the active site, preventing substrate binding and enzyme activity.


What blocks a bonding site in muscle?

Several things do. Neuromuscular-blocking drugs block neuromuscular transmission at the neuromuscular junction causing paralysis. Quaternary ammonium muscle relaxants are quaternary ammonium salts used as drugs for muscle relaxation, most commonly in anesthesia.When a muscle is in a resting state, actin and myosin are separated. To keep actin from binding to the active site on myosin, regulatory proteins block the molecular binding sites. Tropomyosin blocks myosin binding sites on actin molecules, preventing cross-bridge formation, which prevents contraction in a muscle without nervous input.Several things do. Neuromuscular-blocking drugs block neuromuscular transmission at the neuromuscular junction causing paralysis.Quaternary ammonium muscle relaxants are quaternary ammonium salts used as drugs for muscle relaxation, most commonly in anesthesia.Tropomyosin blocks myosin binding sites on actin molecules, preventing cross-bridge formation, which prevents contraction in a muscle without nervous input.


The building blocks for this molecule are?

ANSWER: You are absolutely right: All molecules are made up of tiny atoms. ... This happens because of the formation of bonds between the atoms, which holds them together to make a molecule.


How can the consumption of caffeine help reduce adenosine levels in the body?

Caffeine blocks adenosine receptors in the brain, preventing adenosine from binding to them. This leads to increased alertness and reduced feelings of tiredness.


Which blocks enzyme activity by binding to the site of an enzyme?

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How do repressors affect promoters?

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What blocks HIV binding to host cell receptors?

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What gene specifies a protein that binds to an operatr and physically blocks rna polymerase from binding to a promoter site?

The gene that specifies a protein known as a repressor can bind to the operator region in the DNA, physically blocking RNA polymerase from binding to the promoter site. This mechanism helps regulate gene expression by preventing transcription of the gene.


What happens when a repressor is bound to the operator?

When a repressor is bound to the operator, it physically blocks the RNA polymerase from binding to the promoter region of the gene, preventing transcription. This mechanism effectively shuts down gene expression by inhibiting the initiation of transcription of that specific gene.


What blocks the binding sites on actin?

TROPOMYOSIN MOLECULES ( troponin hold the tropomyosin in place)


Nucleotides are the building blocks of which type of molecule?

Nucleotides are the building blocks of nucleic acids, which include DNA and RNA. They are composed of a phosphate group, a sugar molecule, and a nitrogenous base.