Some cases of hypertrophy are due to genetics, while others are acquired later in life. The genetic form is called hypertrophic cardiomyopathy, whose prevalence is 1/500 individuals. There are several causes, most (if not all) of which are due to defects in the genes that encode certain proteins that control contraction of heart muscle.
Acquired cardiac hypertrophy usually refers to hypertrophy of the ventricles, most commonly the left ventricle. Left ventricular hypertrophy is most commonly due to excess work being placed on the left side of the heart. In the United States, a common source of this excess work is high blood pressure (hypertension).
DNA is the genetic code
genetic modification can be bad because animals and stuff are changed to taste better but im not sure if that is genetic engineering
for the genetic engenerring to take place it needs genetic code like for a car to drive you need to put gas in it. its simple
I don't think inflammation is genetic.
Short Answer is: our understanding of genetic engineering.
Stress on the muscles forces adaptation, or hypertrophy of the muscles to the activity. To continue growth, you must continue to increase the stress of the exercise such as lifting more weights.
It won't. But in the long term the murmur (depending on which valve is affected) will cause hypertrophy of cardiac muscles for the different ventricles/atria. And the hypertrophy will be detected by the ECG because it will change the cardiac axis. Murmurs are easily heard using a stethoscope so and ECG isn't useful. An ECG monitors the electric impulses of the heart muscle - so better to see heart blocks, infarcts, hypertrophy, AF, SVT etc
Increase in the size of an organ caused by an increase in the size of its cells is referred to as hypertrophy. This process can occur in response to increased workload or hormonal stimulation, leading to enhanced function of the organ. Unlike hyperplasia, which involves an increase in the number of cells, hypertrophy focuses on the enlargement of existing cells. Common examples include muscle hypertrophy from strength training and cardiac hypertrophy from increased blood pressure.
Cardiac enlargement is associated with dynamic training. The heart's response to static training is hypertrophy, thickening of the muscle walls of the heart
Cardiac myocytes (heart muscle cells) do not regenerate. They can get bigger (hypertrophy), but new cells are not made under normal circumstances. This means that when you have a heart attack or another injury to the heart and cardiac cells die, they are replaced by fibrous scar tissue which does not contract like normal heart tissue does.
They include: - Muscle hypertrophy (enlargement) - Increased muscular stores of ATP and PC - Increased glycolytic capacity - Cardiac hypertrophy - Other anaerobic training adaptations
Hypertrophy.
The stretching and enlargement of the heart without any additional cells is known as cardiac hypertrophy. This can occur in response to chronic conditions such as high blood pressure or heart disease, leading to the heart muscle thickening and working harder to pump blood efficiently. Over time, untreated hypertrophy can weaken the heart muscle and lead to complications like heart failure.
basically cardiac hypertrophy is when your heart increases in size and blood volume. the wall of the left ventricle doesnt thicken but instead the right one does, increasing the strength potential of its contractions. and then during exercise your chest becomes bigger, which enables thicker muscles being built, and as your right ventricle gets bigger your right pectoral may become bigger than the left one but as you grow your pectorals will be the same length thanks, AKA.
The hypertrophy of the liver was causing the problem.
right ventricle hypertrophy
If afterload increases, cardiac output may decrease, assuming other factors remain constant. This is because the heart has to work harder to eject blood against the higher resistance, potentially leading to reduced stroke volume. Over time, the heart may compensate through hypertrophy, but acute increases in afterload typically result in diminished cardiac performance.