Apoptosis is cell death via shrinkage, whereas oncotic necrosis is cell death via swelling.
The term necrosis was used for cell death before these two different processes (shrinkage and swelling) were discovered. For this reason, it is still sometimes used to refer to both apoptosis and oncotic necrosis. However, necrosis is also sometimes used to mean only cell death via swelling.
To avoid confusion, it is best to use the terms apoptosis and oncotic necrosis (and not just 'necrosis').
by apoptosis and necrosis
necrosis is death of cells with underlying/ or death of cells that cause inflammation. necrosis has to have an underlying injury. ischemia is decreased blood supply to tissues/ cells due to obstruction/ constriction of vasculature - indeed, this is an injury, and can lead to necrosis of cells if not resolved quickly! Note that apoptosis is programmed cell death - i.e. nucleus control is involved, and there is no inflammation - it is clean cell death! this is awesome.
Caspases, also known as cysteine-aspartic proteases are a family or cysteine proteases that play an essential role in apoptosis, necrosis and inflammation. Capsases are essential in cells for apoptosis.
Brain trauma injury can lead to both necrosis and apoptosis of brain cells. Necrosis is often seen in the initial injury phase due to the mechanical damage, while apoptosis may occur in the subsequent days as a result of secondary injury processes such as inflammation and oxidative stress. Both types of cell damage can contribute to the overall brain damage and neurologic deficits following a traumatic brain injury.
Apoptosis-is mass cell death which kills and stops bodily functions and homeostasis to repair deadcells.Cancer-Uncontrolable cell growth or amount.
Unplanned cell death, opposite of apoptosis.
The term for tissue death is either necrosis or apoptosis, depending on the situation. Apoptosis is preprogrammed tissue death, while necrosis is not preprogrammed. An infarct is an area of tissue that has died due to lack of blood supply.
Fat necrosis is one of many types of necrosis. Necrosis is cell death with inflammation (different from apoptosis, which is without inflammation). Fat necrosis occurs in two forms. 1. Traumatic fat necrosis is the result of trauma. Cellular damage to fat-rich organs, such as the breasts, can lead to necrosis of the adipocytes. 2. Fat necrosis as a result of acute haemorrhagic pancreatitis. In this case, damage to the pancrease releases lipolytic enzymes into the blood, causing damage, and eventual necrosis of adipocytes.
Necrosis means that a cell or a group of cells die due to injury (heat, pressure etc), diseases (infections), or other pathological state. The term "infarction" essentially is synonymous with "ischemic necrosis" which means necrosis (cell death) from the lack of blood (ischemia). Ischemia can be caused by the occlusion of the arterial supply or venous drainage for a particular tissue.
Liquefactive necrosis is good for your body, especially your brain (due to the beneficial liquids produced) but coagulative necrosis is bad due to the clumping and coagulation (i.e. clotting) that occurs.Coagulation necrosis is the "acute" necrosis in which the protein fibers become denatured and everything turns into a semi-solid mess of dead tissue. Liquefactive necrosis is a more "chronic" necrosis in which the dead tissue is digested into a liquid which can then be removed by the macrophages.
Necrosis is considered a passive process because it is unregulated and typically results from external factors such as injury or lack of blood supply. In necrosis, cells swell and burst, releasing their contents into the surrounding tissue, which can trigger inflammation and damage nearby healthy cells. This is in contrast to apoptosis, which is a programmed cell death process that is controlled by the cell itself.
It depends on which caspase we are talking about - some of them are involved in cell apoptosis, some in inflammatory processes. So you will either have a decreased apoptotic and necrosis scenario, or decreased inflammatory pathways.