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What neurotransmitter coordinates muscle actions a. acetylcholine b. epinepherine c. endorphin d. serotonin e. GABA?
A, acetylcholine. Acetylcholine is commonly secreted at neuromuscular junctions, the gaps between motor neurons and muscle cells, where it stimulates muscules to contract. At other kinds of junctions, it typically produces and inhibitory post-synaptic potential.
When an action potential arrives at the neuromuscular junction the most immediate response?
is the release of acetylcholine from the motor neuron into the synaptic cleft.
What is the neurotransmitter of the motor neuron?
Alpha-Motor neurons release the neurotransmitter acetylcholine at a synapse called the neuromuscular junction. When the acetylcholine binds to acetylcholine receptors on the muscle fiber, an action potential is propagated along the muscle fiber in both directions.
What is the neurotransmitter of alpha-motor neuron?
Alpha-Motor neurons release the neurotransmitter acetylcholine at a synapse called the neuromuscular junction. When the acetylcholine binds to acetylcholine receptors on the muscle fiber, an action potential is propagated along the muscle fiber in both directions.
How does botulism toxin affect the neuromuscular junction?
It affects the axon terminal, by blocking the release of acetylcholine , thus preventing an action potential from occurring.
What potential motor end plate to synapse to acetylcholine synaptic cleft to special receptors on the skeletal muscle to action potential across the muscle travels down t-tubule to the what?
This is the neuromuscular junction. The action potential travels down the presynaptic motor neuron, releasing acetylcholine (ACh), which cross the synapse (neuromuscular junction), to bind to receptors on the end plate of muscle fibers, which induces depolarization, causing the muscle to contract.
What describes a critical event that occurs at the neuromuscular junction?
A critical event that occurs at the neuromuscular junction is the release of acetylcholine from the motor neuron's axon terminal. This neurotransmitter binds to receptors on the muscle membrane, leading to depolarization of the muscle cell and generation of an action potential, initiating muscle contraction. Dysfunctions at the neuromuscular junction can lead to diseases like myasthenia gravis.
What is the difference between depolarizing and non-depolarizing drugs?
Depolarizing neuromuscular blocking drugs, like succinylcholine, cause initial depolarization of the muscle cell membrane followed by prolonged depolarization. Non-depolarizing drugs, like rocuronium, block the action of acetylcholine at the neuromuscular junction without causing depolarization.
Describe the components of the neuromuscular junction?
The neuromuscular junction consists of the motor neuron terminal, synaptic cleft, and motor end plate on the muscle fiber. When an action potential reaches the motor neuron terminal, it triggers the release of acetylcholine into the synaptic cleft. Acetylcholine then binds to receptors on the motor end plate, leading to muscle contraction.
What would happen if acetylcholine was released into a neuromuscular junction?
It attaches to its receptor, and binds. Causes another action potential, (calcium released) and it goes to the T-tubule of the Sarcoplasmic reticulum, and attaches to troponin, which moves the tropomyosin, so the myosin and actin and attach, and cause a muscle contraction.
A folded region of the sarcolemma at the neuromuscular junction?
The folded region of the sarcolemma at the neuromuscular junction is called the motor end plate. It is responsible for receiving signals from the motor neuron and initiating muscle contraction. The motor end plate is rich in acetylcholine receptors, which play a key role in neurotransmission at the neuromuscular junction.
Chemical released to stimulate muscle contraction?
Acetylcholine is the primary neurotransmitter released by motor neurons at the neuromuscular junction to stimulate muscle contraction. It binds to receptors on the muscle cell membrane, initiating a series of events that ultimately lead to muscle contraction.
