It attaches to its receptor, and binds.
Causes another action potential, (calcium released) and it goes to the T-tubule of the Sarcoplasmic reticulum, and attaches to troponin, which moves the tropomyosin, so the myosin and actin and attach, and cause a muscle contraction.
Aluminum particles can stay airborne for several days when released into air
it is released in the atmosphere
Most neurotransmitters are removed by being taken up by the presynaptic or postsynaptic neurones however acetylcholine is the prime exeption to this as is actually destroyed in the synaptic cleft by the enzyme acetylcholinesterase. The reason this must happen is that otherwise the neurotransmitter would be left in the cleft where it would continue to evoke a response in the postsynaptic cell for longer than it should. For this reason reuptake and catabolic enzymes are often the targets of drugs gieven to treat neurological disorders. Another possible problem is wastage, if the neurotransmitter is left in the cleft it may difuse away and be wasted giving the presynaptic neurone more work to do creating more.
If acetylcholinesterase were absent, acetylcholine would accumulate in the synaptic cleft, leading to prolonged stimulation of postsynaptic receptors. This could result in continuous muscle contraction, paralysis, or overstimulation of the nervous system, potentially causing symptoms such as muscle spasms and respiratory failure. Ultimately, the absence of this enzyme disrupts normal neurotransmission and can be life-threatening.
Carbon dioxide is released from water at high temperature.
When acetylcholine does not bind to muscle cells, it prevents the activation of nicotinic acetylcholine receptors at the neuromuscular junction. This failure to activate the receptors inhibits the influx of sodium ions, which is necessary for generating an action potential in the muscle fiber. Consequently, muscle contraction cannot occur, leading to muscle weakness or paralysis. This can happen in various conditions, such as myasthenia gravis or in the presence of certain toxins.
Haunted Junction happened in 1997.
If the acetylcholine receptor was destroyed that the effector cells cannot respond or detect the neurotransmitter, resulting in muscle paralysis.
nothing
The general name for a chemical released when a neural signal reaches a neural ending is neurotransmitter.Which specific neurotransmitter is released depends on exactly which neuron has fired; some instances of specific neurotransmitters are: adrenaline (epinephrine), glutamate, GABA (gamma-aminobutyric acid), acetylcholine, dopamine, serotonin, & melatonin.With respect specifically to a neuromuscular junction, and with minor corrections, another contributor wrote:When the nerve impulse reaches the nerve ending calcium is allowed into the cell and the synaptic vesicles in the motor neuron fuse to the bottom of the axon terminal of the motor neuron where they release ACh(Acetylcholine) into the synaptic cleft. So in this particular instance, Acetylcholine is your answer.
Operation Junction City happened on 1967-05-14.
Tapuah junction stabbing happened on 2010-02-10.
Dalton Junction rail crash happened in 1869.
Mehola Junction bombing happened on 1993-04-16.
Coppenhall Junction rail crash happened in 1962.
Barnsley Junction rail accident happened in 1885.
Yagur Junction bombing happened on 2002-04-10.