well we know during severe hypertension narrowing of arterioles and arteries takes place it is common
because of narrowed arteries provides resistance to blood flow hence its flow to lower extremeties ceases and glomerular filtration doesnot occur properly
as filtration decreases the sodium ions and chloride ions are not filtered and remained in the blood we can note that there is a decreased amount of sodium ions in distal tubule of nephron so there is insufficient of ions to transport across the macula densa cells of juxtraglomerular apparatus which are very close to distal tubule of nephron thus on compensation these cells triggers to release renin, after several pathway in the body like renin cleaves a peptide called angiotensinogen -->angiotensin-1---->angiotensin-2(A-2), so this A-2 is useful for glomerular filtration by constricting efferent arteriole more and more than afferent arteriole in order to increase the amount of blood to remain in glomerulus for filtration,,, people might expect that here the glomerular filtration occurs and more sodium ions are excreted but how long ? no it happens for short time because A-2 is responsible for constriction of important arteries and arterioles mainly happens when hypertension is severe thus blood flow decreases and even sufficent blood doesnt reaches to afferent arteriole of glomerulus thus filtration ceases and sodium and chloride ions are not excreted
beside A-2 is also responsible for formation of aldosterone whose main function is retention of water sodium and chloride ions in distal tubule after filtration but the potassium ions are excreted
silly !!!!!!!this stupid A-2 is also responsible for antidiuretic hormone from pituatary gland thus totally retention retention retention of water sodiium and chloride ;;;;;;he he he
ok so the answer is NO, DURING HYPERTENSION the sodium doesnot excreted in large quantities nevertheless less than the normal
so this is the physiology on the view of this, we can probably say the answer : NO
"of my level best i explained if any thing wrong please reply"
if any thing ambigious please reply
Yes, diuretics are medications specifically designed to increase urine secretion. They promote the excretion of water and electrolytes, primarily sodium, from the kidneys, leading to increased urine production. Diuretics are commonly used to manage conditions such as hypertension, heart failure, and edema. By enhancing urine output, they help reduce fluid overload in the body.
Yes, indapamide is a diuretic. It belongs to the thiazide-like class of diuretics and is primarily used to treat hypertension and edema. Indapamide works by inhibiting sodium reabsorption in the kidneys, leading to increased urine output and reduced blood pressure.
If the reabsorption of sodium by the renal tubule is inhibited, it can lead to increased sodium excretion in the urine, which can result in higher urine output (diuresis). This can lead to loss of water and electrolytes, potentially causing dehydration, electrolyte imbalances, and alterations in blood pressure.
In acute glomerulonephritis, urine output is often decreased, a condition known as oliguria. This occurs due to inflammation and damage to the glomeruli, which impairs their ability to filter blood effectively. As a result, the kidneys may retain water and electrolytes, leading to fluid retention and potentially contributing to edema and hypertension. In some cases, urine may also appear dark or smoky due to the presence of blood and protein.
Thiazide diuretics primarily act on the distal convoluted tubule of the nephron. They inhibit the sodium-chloride symporter, leading to reduced reabsorption of sodium and chloride ions. This action increases urine output and decreases fluid retention, making thiazides effective in treating conditions like hypertension and edema.
Caffeine: Acts as a diuretic by increasing blood flow to the kidneys. Alcohol: Inhibits the release of vasopressin, a hormone that helps the body retain water. Certain medications: Such as diuretics, used to treat conditions like hypertension and heart failure. High-sodium foods: Can increase urine output by promoting water excretion.
Furosemide belongs to the category of loop diuretics. It is primarily used to treat conditions such as edema associated with heart failure, liver cirrhosis, and kidney disorders, as well as hypertension. By inhibiting the reabsorption of sodium and chloride in the loop of Henle in the kidneys, it promotes increased urine output.
If urine output is greater than intake a pressure differential will exist between inside and outside the urethra and as a result you will urinate your internal organs out.
Urine contain sodium from sodium chloride.
Bumetanide is a potent loop diuretic that increases urine output by inhibiting sodium and chloride reabsorption in the ascending loop of Henle, leading to increased excretion of water, sodium, and potassium. Patients receiving Bumetanide typically experience a significant increase in urine output, which can be beneficial in managing conditions like heart failure and edema. However, close monitoring is essential, as excessive diuresis can lead to dehydration and electrolyte imbalances.
There are many things that affect an animal's urine output such as food intake. Beverage intake also affects an animal's urine output.
Coffee increases urine output primarily due to its caffeine content, which is a diuretic. Caffeine stimulates the kidneys to filter blood more efficiently, leading to increased urine production. Additionally, coffee's hydration properties can prompt the bladder to fill more quickly, further contributing to the urge to urinate. Overall, these effects combine to result in increased urine output after coffee consumption.