0
well we know during severe hypertension narrowing of arterioles and arteries takes place it is common
because of narrowed arteries provides resistance to blood flow hence its flow to lower extremeties ceases and glomerular filtration doesnot occur properly
as filtration decreases the sodium ions and chloride ions are not filtered and remained in the blood we can note that there is a decreased amount of sodium ions in distal tubule of nephron so there is insufficient of ions to transport across the macula densa cells of juxtraglomerular apparatus which are very close to distal tubule of nephron thus on compensation these cells triggers to release renin, after several pathway in the body like renin cleaves a peptide called angiotensinogen -->angiotensin-1---->angiotensin-2(A-2), so this A-2 is useful for glomerular filtration by constricting efferent arteriole more and more than afferent arteriole in order to increase the amount of blood to remain in glomerulus for filtration,,, people might expect that here the glomerular filtration occurs and more sodium ions are excreted but how long ? no it happens for short time because A-2 is responsible for constriction of important arteries and arterioles mainly happens when hypertension is severe thus blood flow decreases and even sufficent blood doesnt reaches to afferent arteriole of glomerulus thus filtration ceases and sodium and chloride ions are not excreted
beside A-2 is also responsible for formation of aldosterone whose main function is retention of water sodium and chloride ions in distal tubule after filtration but the potassium ions are excreted
silly !!!!!!!this stupid A-2 is also responsible for antidiuretic hormone from pituatary gland thus totally retention retention retention of water sodiium and chloride ;;;;;;he he he
ok so the answer is NO, DURING HYPERTENSION the sodium doesnot excreted in large quantities nevertheless less than the normal
so this is the physiology on the view of this, we can probably say the answer : NO
"of my level best i explained if any thing wrong please reply"
if any thing ambigious please reply
What else can I help you with?
Are diuretics administerd to increase urine secretion?
Yes, diuretics are medications specifically designed to increase urine secretion. They promote the excretion of water and electrolytes, primarily sodium, from the kidneys, leading to increased urine production. Diuretics are commonly used to manage conditions such as hypertension, heart failure, and edema. By enhancing urine output, they help reduce fluid overload in the body.
Is indapamide a diruetic?
Yes, indapamide is a diuretic. It belongs to the thiazide-like class of diuretics and is primarily used to treat hypertension and edema. Indapamide works by inhibiting sodium reabsorption in the kidneys, leading to increased urine output and reduced blood pressure.
What happens if the reabsorption of sodium by the renal tubule is inhibited?
If the reabsorption of sodium by the renal tubule is inhibited, it can lead to increased sodium excretion in the urine, which can result in higher urine output (diuresis). This can lead to loss of water and electrolytes, potentially causing dehydration, electrolyte imbalances, and alterations in blood pressure.
What part of the nephron do thiazide diuretics work on?
Thiazide diuretics primarily act on the distal convoluted tubule of the nephron. They inhibit the sodium-chloride symporter, leading to reduced reabsorption of sodium and chloride ions. This action increases urine output and decreases fluid retention, making thiazides effective in treating conditions like hypertension and edema.
List four substance that increase urine production?
Caffeine: Acts as a diuretic by increasing blood flow to the kidneys. Alcohol: Inhibits the release of vasopressin, a hormone that helps the body retain water. Certain medications: Such as diuretics, used to treat conditions like hypertension and heart failure. High-sodium foods: Can increase urine output by promoting water excretion.
What category does furosemide belong to?
Furosemide belongs to the category of loop diuretics. It is primarily used to treat conditions such as edema associated with heart failure, liver cirrhosis, and kidney disorders, as well as hypertension. By inhibiting the reabsorption of sodium and chloride in the loop of Henle in the kidneys, it promotes increased urine output.
What happens if urine output is greater than intake?
If urine output is greater than intake a pressure differential will exist between inside and outside the urethra and as a result you will urinate your internal organs out.
Is sodium chloride in urine?
Urine contain sodium from sodium chloride.
What factors affect urine output?
There are many things that affect an animal's urine output such as food intake. Beverage intake also affects an animal's urine output.
Normal daily urine output in a male?
For a male OR female, drinking about 2 liters of fluid a day, the normal urine output should be 800 to 2,000 milliliters a day. Urine output below 500 milliliters a day is considered a low urine output and is a sign of disease.
What is the result if a few drops of sodium hydroxide solution and copper textraoxosulphate were added to a sample of urine?
If sodium hydroxide solution and copper tetraoxosulphate were added to urine, a color change might occur. This reaction produces a blue color due to the formation of a complex between copper ions and proteins in urine. This test can be used to detect the presence of proteins in the urine.
How does dehydration change urine composition and urine output?
Dehydration leads to a decrease in urine output as the body conserves water to maintain hydration levels. As a result, urine becomes more concentrated, exhibiting higher levels of solutes such as urea, creatinine, and electrolytes. This change in composition can also cause urine to appear darker in color. Overall, dehydration prompts the kidneys to reabsorb more water, reducing the volume of urine produced.
