Cardiac output (CO) is determined by the heart rate (HR) and the volume of blood pumped by each beat (stroke volume - SV). Mathematically, cardiac output can be represented by the equation:
CO = HR x SV
As such, if total cardiac output falls as a result of decreased stroke volume, the heart rate can increase to keep the total cardiac output normal, to a certain extent.
Stroke volume is more complicated; it is determined by many different factors, including preload, afterload, competence of the atrioventricular valves, ventricular cavity size, and the strength of the squeeze of the cardiac muscle, amongst others. Any change in one of these factors requires a compensation in one or more of the others to maintain cardiac output.
--Sympathetic nervous system stimulation (inc. catecholemines) --RAAS activation --Myocardial hypertrophy --Increased CO, with a rise in left ventricular end-diastolic pressure
repolariztion of the cardiac cells. ie after they have depolarized so quickly it takes a while for them to get back to a normal state
Increase in heart rate as Cardiac Output = Heart rate x Stroke volume. As SV will be decreased, HR increases to compensate.
Blood pressure (BP) is a late sign of shock in pediatric patients because children often have compensatory mechanisms that maintain BP until they are significantly compromised. In the early stages of shock, children can increase their heart rate and peripheral vascular resistance to preserve organ perfusion, leading to normal BP despite critical internal changes. As shock progresses and compensatory mechanisms fail, BP drops, indicating severe cardiovascular compromise. Thus, reliance on BP alone can delay recognition and treatment of shock in pediatric patients.
Shock can significantly impact the heart by causing decreased blood flow and oxygen delivery to cardiac tissues, leading to potential damage. In cases of hypovolemic shock, for example, reduced blood volume results in lower cardiac output and can precipitate arrhythmias. Additionally, the body’s stress response during shock can lead to increased heart rate and contractility, but prolonged shock may overwhelm these compensatory mechanisms, resulting in heart failure. Overall, timely medical intervention is crucial to mitigate these effects and restore normal heart function.
A compensatory pause on an ECG refers to a pause in heart rhythm that follows a premature beat or extra heartbeat. This pause allows the heart to reset and resume its normal rhythm. It is a normal mechanism of the heart's electrical system to ensure effective pumping.
Cardiac silhouette is prominent with a left ventricular configuration.
0.8 seconds
12%.
If the cardiac size is within the upper limits of normal, it means that the heart is still a normal size, but is on the larger size of all normal hearts. There is a range for how big the heart is allowed to be.
pottasium and sodium
defibrillators