No, post-ganglionic sympathetic fibers mostly release norepinephrine.
But they do release some ACh to the sweat glands, which causes concurrent vasodilation of surrounding blood vessels (paired with release of bradykinin from sweat glands).
Preganglionic sympathetic fibers trigger the release of acetylcholine at the synapse with postganglionic neurons in the sympathetic ganglia. This neurotransmitter binds to nicotinic acetylcholine receptors on the postganglionic neuron, which then propagate the signal to release norepinephrine at the target tissue.
The autonomic nervous system fibers that release norepinephrine are primarily the postganglionic sympathetic fibers. These fibers originate in the sympathetic ganglia and project to various target organs, where they release norepinephrine to mediate the "fight or flight" response. In contrast, the preganglionic sympathetic fibers release acetylcholine.
No, parasympathetic postganglionic fibers do not come from adrenergic fibers. Parasympathetic postganglionic fibers release acetylcholine as their neurotransmitter, whereas adrenergic fibers release norepinephrine.
Postganglionic efferent fibers terminate at effector organs, which include smooth muscles, cardiac muscles, and glands. These fibers are part of the autonomic nervous system, which is divided into the sympathetic and parasympathetic systems. In the sympathetic system, they typically release norepinephrine, while in the parasympathetic system, they primarily release acetylcholine to exert their effects on target tissues.
Preganglionic fibers for the SNS release ACh; so a drug that stimulates ACh receptors would stimulate the postganglionic fibers of sypathetic nerves, resulting in increased sympathetic activity
Norepinephrine is mainly secreted by the adrenal medulla and the postganglionic fibers of the sympathetic nervous system, which are also known as adrenergic fibers. These fibers release norepinephrine in response to stress or arousal, helping to initiate the body's fight or flight response.
Parasympathetic
it would be postganglionic sympathetic fibers
Andrenergic fibers release Norepinephrine and Cholinergic fibers release Acetylcholine.
Sympathetic postganglionic fibers end in effector organs such as smooth muscles, cardiac muscles, and glands. They innervate these structures to modulate their activity in response to stress or stimulation of the sympathetic nervous system.
Yes, when stimulated the sympathetic postganglionic fibers release norepinepherine (20%) and epinepherine (80%).
In some muscle tissue acetylcholine causes vaso-dilation, but not all. Norepinephrine is the opposite competor/effector of acetylcholine. Acetylcholine is present in all preganglionic fibers, both parasympathetic and sympathetic. Acetylcholine is present in postganglionic parasympatic fibers, where norepinephrine is present in the postganglionic sympathetic fibers. In some tissues acetylcholine causes constriction. Can also reduce heart rate vi the vagus nerve. Acetylcholine is the only neurotransmitter used in the somatic nervous system! Acetylcholine can effect vasodilation by several mechanisms, including activation of endothelial nitric oxide (NO) synthase and prostaglandin (PG) production. In human skin, exogenous Acetylcholine increases both skin blood flow and bioavailable NO levels, but the relative increase is much greater in skin blood flow than NO. So this may lead us to speculate that acetylcholine may dilate cutaneous blood vessels through PGs, as well as NO. In some muscle tissue acetylcholine causes vaso-dilation, but not all. Norepinephrine is the opposite competor/effector of acetylcholine. Acetylcholine is present in all preganglionic fibers, both parasympathetic and sympathetic. Acetylcholine is present in postganglionic parasympatic fibers, where norepinephrine is present in the postganglionic sympathetic fibers. In some tissues acetylcholine causes constriction. Can also reduce heart rate vi the vagus nerve. Acetylcholine is the only neurotransmitter used in the somatic nervous system! Acetylcholine can effect vasodilation by several mechanisms, including activation of endothelial nitric oxide (NO) synthase and prostaglandin (PG) production. In human skin, exogenous Acetylcholine increases both skin blood flow and bioavailable NO levels, but the relative increase is much greater in skin blood flow than NO. So this may lead us to speculate that acetylcholine may dilate cutaneous blood vessels through PGs, as well as NO.