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In some muscle tissue acetylcholine causes vaso-dilation, but not all. Norepinephrine is the opposite competor/effector of acetylcholine. Acetylcholine is present in all preganglionic fibers, both parasympathetic and sympathetic. Acetylcholine is present in postganglionic parasympatic fibers, where norepinephrine is present in the postganglionic sympathetic fibers. In some tissues acetylcholine causes constriction. Can also reduce heart rate vi the vagus nerve. Acetylcholine is the only neurotransmitter used in the somatic nervous system! Acetylcholine can effect vasodilation by several mechanisms, including activation of endothelial nitric oxide (NO) synthase and prostaglandin (PG) production. In human skin, exogenous Acetylcholine increases both skin blood flow and bioavailable NO levels, but the relative increase is much greater in skin blood flow than NO. So this may lead us to speculate that acetylcholine may dilate cutaneous blood vessels through PGs, as well as NO. In some muscle tissue acetylcholine causes vaso-dilation, but not all. Norepinephrine is the opposite competor/effector of acetylcholine. Acetylcholine is present in all preganglionic fibers, both parasympathetic and sympathetic. Acetylcholine is present in postganglionic parasympatic fibers, where norepinephrine is present in the postganglionic sympathetic fibers. In some tissues acetylcholine causes constriction. Can also reduce heart rate vi the vagus nerve. Acetylcholine is the only neurotransmitter used in the somatic nervous system! Acetylcholine can effect vasodilation by several mechanisms, including activation of endothelial nitric oxide (NO) synthase and prostaglandin (PG) production. In human skin, exogenous Acetylcholine increases both skin blood flow and bioavailable NO levels, but the relative increase is much greater in skin blood flow than NO. So this may lead us to speculate that acetylcholine may dilate cutaneous blood vessels through PGs, as well as NO.
What else can I help you with?
What causes vaodilation?
Corticotrpin is the harmone which increases vasodilation in our body. alcohol causes vasodilation
Does sympathetic activity cause vasodilation?
No, sympathetic activity typically causes vasoconstriction, not vasodilation.
Does sympathetic stimulation cause vasodilation of which arterioles?
Sympathetic stimulation causes vasoconstriction of most arterioles in the body, except for arterioles in skeletal muscle and the heart where it causes vasodilation.
The enzyme cholinesterase causes acetylcholine to what?
Decompose
Do the post-ganglionic sympathetic fibers release acetylcholine?
No, post-ganglionic sympathetic fibers mostly release norepinephrine. But they do release some ACh to the sweat glands, which causes concurrent vasodilation of surrounding blood vessels (paired with release of bradykinin from sweat glands).
Does histamine cause blood vessels to contract?
No it causes them to dilate (vasodilation)
What chemical into the muscle cell membrane causes a temporary rush?
Acetylcholine (ACh)
What is relaxation of the smooth muscle in the artery wall which causes the artery wall to become larger?
Vasodilation
Botulin poisoning from improperly canned food causes paralysis by blocking the release of what?
Acetylcholine
Binding of the Acetylcholine to receptors causes a by opening what channels that permit both potassium and sodium to permeate the membrane?
Binding of acetylcholine to nicotinic acetylcholine receptors opens ion channels that allow both sodium and potassium ions to permeate the membrane. This causes depolarization of the membrane potential, leading to an excitatory response in the cell.
What neurotransmitter causes muscle movement?
Acetylcholine is the primary neurotransmitter responsible for muscle movement. It is released from motor neurons at the neuromuscular junction to stimulate muscle contraction.
Cholinesterase causes acetylcholine to do what?
Cholinesterase breaks down acetylcholine into choline and acetic acid. This process helps to quickly terminate the signal transmission at cholinergic synapses, preventing overstimulation and allowing the synapse to reset for the next signal.
