Peripheral resistance can be increased by an increase in blood volume and the constricting of blood vessels.
An increase in the sympathetic nervous system can lead to an increase in stroke volume. This is because the sympathetic nervous system stimulates the heart to beat faster and with more force, resulting in more blood being pumped out with each heartbeat.
Cardiac Output is the product of Stroke Volume and Hear Rate. Increase in either one will increase in Cardiac OutPut. THe increase in HR could be due to decrease of Parasympatheic Drive, Incresae in Sympathetic Drive. (norepiphrine increase HR) Peripheral REsistance decrease because of vasodilation during exercise in the working musclse because of vasodilator metabolites (adenosine, K+) inhibit Norepinphrine release locally. I think the increase in pulse and blood pressure duing exercise is to meet the oxygen demand of the working tissue. In unfir person you can see that they acheive that mostly by increasing heart rate. However when you see the fit person doing the same workload, they might have lesser increase of HR compare to the unfit person because they have a stroger stroke volume. I hope this helps
This will result in reduced pre-load for the left ventricle, thereby reducing the effective cardiac output into the systemic circulation. The consequence of this is reduced blood pressure and could lead to cardiogenic shock if it is severe enough. Decreased venous return can occur as a result of a number of situations. Severe haemorrhage, Pericarditis, peripheral vasodilation as a result of neurogenic shock to name a few. Each cause reduced venous return for different reasons - either through reduced blood volume (haemorrhage), compression of the heart chambers (pericarditis, or any situation where fliud or exudate builds up in the pericardium), or some form of trauma or lesion to the spinal cord - specifically affecting the sympathetic tract - which disrupts sympathetic nervous impulses to the peripheral blood vessels, causing widespread vasodilation below the site of the lesion/trauma (neurogenic shock).
stimulation of the sympathetic nervous system and the release of adrenaline, which causes vasoconstriction and an increase in heart rate. Other factors that can increase arterial blood pressure include stress, physical activity, and certain medications.
The resistance offered by the peripheral circulation is known as the systemic vascular resistance (SVR). Vasoconstriction (i.e., decrease in blood vessel diameter) increases SVR, whereas vasodilation (increase in diameter) decreases SVR. this constriction and dialation decreases or increases the volume in which the vessles can potensially hold. the blood pressure is subsequantly altered as more or less vascular space is provided.
Studies have found that respiration can alter the blood volume levels to the fingertips. When inhaling the blood volume levels can increase whilst the blood levels can decrease in the fingertips when exhaling. This is because respiration causes fluctuations in arterial blood pressure and heart rate.
Heart rate increases with sympathetic nervous system. There is increase in stroke volume and cardiac output. With stimulation of vagus nerve or parasympathetic nervous system, You have decrease in heart rate. There is decrease the stroke volume and cardiac output.
increased contractility
parasympathatic N.S tends to decrease heart rate , giving more time for diastolic filling and thus increasing the EDV , and increasing the SV according to Frank-starlling law. but this doesn't imply an increase in the cardiac output , because i depend on the heart rate too(which was declined)
End-diastolic volume (EDV), total peripheral resistance (TPR), and contractility.
Ofcourse, the volume will increase Ofcourse, the volume will increase