Acetylcholine (ACh) is a neurotransmitter that plays a crucial role in muscle movement by transmitting signals from motor neurons to muscle fibers at the neuromuscular junction. When ACh is released, it binds to receptors on the muscle cell membrane, leading to depolarization and the initiation of an action potential. This action potential triggers a series of events that result in muscle contraction. Thus, ACh is essential for coordinating voluntary muscle movements.
ACh (acetylcholine) binds to receptors at the NMJ (neuromuscular junction) to induce contraction of muscle.
The destruction of acetylcholine (ACh) receptors on the sarcolemma impairs muscle contraction by preventing effective communication between motor neurons and muscle fibers. When ACh is released from motor neurons, it binds to these receptors to initiate the depolarization of the muscle cell membrane, leading to contraction. Without sufficient ACh receptors, the muscle fibers cannot respond adequately to the neurotransmitter, resulting in weakened or absent muscle contractions. This is a characteristic feature of conditions like myasthenia gravis.
ACh esterase, which breaks ACh down into an acetyl group and a choline group. This is then taken up by the neurone and some is reformed into ACh.
acetylcholine (ACh)
The chemical used at the junction with a skeletal muscle is acetylcholine (ACh). It is released from the motor neuron at the neuromuscular junction and binds to receptors on the muscle fiber's membrane, triggering muscle contraction. This process is essential for voluntary movement and is part of the overall neuromuscular signaling mechanism.
Acetylcholine (ACh)
Chemical A would be the best muscle relaxant. Since the muscle relies on ACH as a stimulant, this chemical would inhibit communication between the muscle cell and the neuron thus making it not contract. While Chemical B would cause further contraction of the muscle since calcium is needed to help the myosin head to attach to the actin filliment which creates muscle contraction.
The purpose of the muscle cell is movement, support, help the heart pump and to heart and warmth\ by H.L
Botulin is best described as a neurotoxin that inhibits the release of acetylcholine (ACh) at the neuromuscular junction, leading to muscle paralysis.
Cholinergic fibers release acetylcholine (ACh) at their synapses to transmit signals to target cells, including muscle cells, gland cells, and other neurons. This neurotransmitter is vital in the functioning of the parasympathetic nervous system and at neuromuscular junctions for muscle contraction.
Acetylcholine is the neurotransmitter at the neuromuscular junction.
If enough ACh is released, the sarcolemma at that point becomes temorarily more permeable to sodium ions, which rush into the muscle cell and to potassium ions which diffuse out of the cell. Although more sodium ions enter than posassium ions leave.