Need more information. Are you a diabetic (if so type 1 or type 2), are you on insulin, are you on medication?
When your blood sugar level drops you are hypoglycemic. You do not need to be a diabetic to have this happen, although it does happen frequently to diabetics. You may feel shaky, nervous, your heart may begin to pound, you may become pale. At this point you need to take a fast acting sugar such as a glucose tab (sugar tablet that you eat) a soda (coke), fruit juice with some added sugar. Wait fifteen minutes and take more if you still are feeling low. After you have stabilized then take about
2 oz. of protein (peanut butter is good). The sugar will bring your blood sugar up within a normal range and then you need the protein to keep you there until your next meal. DO NOT eat chocolate, many have been told to eat a candy bar when having a low sugar reaction but chocolate has fat in it and fat will actually prevent the sugar from entering your blood stream quickly. You need a sugar that will get into your bloodstream quickly.
Yes it does.
It is lowered by insulin, which is produced in the beta cells of the pancreas in an area called the Islet of Langerhans. Blood glucose is also suppressed by somatostatin which is produced in the delta cells of the pancreas.
Insulin causes the glucose in your blood to enter the cells for energy. It does not cause the liver to change glucose into anything. Your liver does, however, store extra sugar in the form of glucagon.
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An increase in blood sugar levels cause the release of the hormone insulin by the pancreas. Insulin then lowers this blood sugar level restoring it to original non-lethal blood glucose levels.
The main hormone that signals the release of glucose into the blood is Glucagon, however, it is used in conjunction with other hormones which also cause a release of glucose into the blood. These are somatostatin, Adrenaline (epinephrine for Americans), Cortisol and ACTH.
Cells located in the pancreas that produces insulin. Insulin controls the amount of glucose in the blood and, when glucose levels spike, cause certain cells to 'suck up' the access glucose and store it.
Insulin takes glucose from the blood and converts it glycogen that can be stored in the liver and muscles.Insulin can increase blood pressure in a reaction called metabolic syndrome. It decreases glucose and developing studies show that developing diabetes may increase calcium levels which can affect artery calcium deposits. These deposits are associated with high blood pressure.
Insulin enables the sugar to get out of the blood and into the cells where it is needed for the cells to function. If you don't have insulin because the pancreas is not producing it (as in diabetes) then the sugar will not be able to get into the cells, therefore there will be a high concentration in the blood. On the other hand, if you are diabetic and you take too much insulin, then too much sugar will go into the cells and there will not be enough sugar left in the blood.
High blood sugar (glucose) levels in the blood are the number one reaction to a decrease in insulin secretion.However, lack of insulin secretion over time can also contribute to an individual obtaining Diabetes.Therecan also be spillage of keytones into the urine (which disrupts pH levels in the blood) from lack of insulin. These keytones can contribute to ketoacidosis which can cause coma or even death.Excessive urination and thirst are also effects of lack of insulin secretion due to the raise of blood sugar.
Increased blood sugar will cause insulin production to rise and the cells will store the excess sugar, increased blood pressure causes a slower heart rate and decrease in oxygen levels will increase red blood cell production.
Insulin is secreted when blood sugar rises. It works by stimulating receptors on tissue cells to escort glucose into the cells. It all suppresses glucagon, hormone-sensitive lipase (a hormone responsible for mobilizing and using fat stores) and suppresses the appetite centers in the brain. Glucagon is secreted when blood sugar is low. It triggers gluconeogenesis in the liver, which means that fats and proteins are converted into glucose and released into the blood. It also is responsible for mobilizing glycogen stores, suppresses insulin, and mobilizes fat stores. So glucagon and insulin not only do opposite things, they are antagonistic to each other, meaning when one is high the other one is low. They do not technically work "together". High levels of insulin are common in those who consume excessive calories and excessive carbohydrate foods. For those who are sensitive to the effects of insulin, this can cause reactive hypoglycemia and obesity.