Serum ACE levels are elevated in approximately 80-90% of patients with active sarcoidosis
The primary source of ACE is the endothelium of the lung. ACE activity is increased in sarcoidosis, a systemic granulomatous disease that commonly affects the lungs. In sarcoidosis, ACE is thought to be produced by epithelioid cells and macrophages of the granuloma.Currently, it appears that ACE activity reflects the severity of sarcoidosis: 68% positivity in those with stage I sarcoidosis, 86% in stage II sarcoidosis, and 91% in stage III sarcoidosis. Serum ACE also appears to reflect the activity of the disease; there is a dramatic decrease in enzyme activity in some patients receiving prednisone.Other conditions such as Gaucher disease, leprosy, untreated hyperthyroidism, psoriasis, premature infants with respiratory distress syndrome, adults with amyloidosis, and histoplasmosis have been associated with increased levels of ACE.
Serum Angiotensin-Converting Enzyme (ACE) is an enzyme involved in regulating blood pressure by converting angiotensin I to angiotensin II, a potent vasoconstrictor. It is also involved in the degradation of bradykinin, a vasodilator. Measurement of serum ACE levels can be used in diagnosing and monitoring certain diseases like sarcoidosis.
The ACE test is used primarily to detect and monitor the clinical course of sarcoidosis
An elevated ACE (angiotensin-converting enzyme) level in the cerebrospinal fluid (CSF) can be seen in conditions such as sarcoidosis, a systemic inflammatory disorder that can involve the central nervous system. It is an indicator of increased immune activity in the CNS and can help in the diagnosis and monitoring of neurological involvement in sarcoidosis.
An Angiotensin-Converting Enzyme (ACE) test measures the levels of ACE in the blood. ACE is an enzyme that plays a role in regulating blood pressure and fluid balance in the body. Abnormal levels of ACE can be associated with conditions such as sarcoidosis and other inflammatory diseases.
Low levels of angiotensin-converting enzyme (ACE) can be caused by genetic factors, certain medications like ACE inhibitors, or conditions such as sarcoidosis which can lead to decreased production or inactivation of the enzyme.
It could. If its a diabetic patient who has raised serum postassium due to diabetic nephropathy then ace inhibitor can improve his diabetic nephropathy leading to hypokalemia.... BUT it DOESNT cause hypokalemia directly... instead it leads to hyperkalemia...
It's an ACE inhibitor for hypertension. Inhibition of ACE results in a decrease in plasma angiotensin II, leading to decreased vasoconstriction and a small decrease in aldosterone secretion and plasma aldosterone concentrations. Although the decrease in aldosterone is small, it can result in small increases in serum potassium. Slight increases in serum potassium have been observed in some hypertensive patients treated with benazepril alone. Essentially no change in mean serum potassium was seen in patients treated with benazepril and a thiazide diuretic .
Angitensin-converting enzyme (ACE) inhibitors can increase creatinine levels by causing an increase in serum potassium.
No lisinopril is an ACE inhibitor and does not affect the prostate
Angiotensin-converting enzyme (ACE) tests measure the levels of ACE in the blood. Elevated ACE levels can indicate conditions like sarcoidosis, a lung condition that causes inflammation, or other granulomatous disorders. Decreased levels can occur in conditions like cirrhosis or Wilson's disease.
If you mean the Axis of the Earth, yes it did. By 6 degrees.