Tetanus affects motor neurons in the central nervous system, particularly those that control muscle movement. The tetanus toxin interferes with neurotransmission at the neuromuscular junction, leading to muscle stiffness and spasms.
No, A B toxin is not a membrane disrupting toxin. It consists of two subunits: the A subunit, which is the active toxic component, and the B subunit, which is responsible for binding to cell surface receptors and facilitating the entry of the A subunit into the cell.
The most likely structure of the plant cell to be affected by a toxin would be the cell membrane or the chloroplasts. Toxins can disrupt the cell membrane, leading to cell damage. Additionally, toxins can interfere with chloroplast function, affecting photosynthesis and overall plant health.
The function of a cell membrane is to allow certain things in, such as nutrients, ans out such as toxin wastes out of the cell. This allows the cell to get what it needs in order to function. If a cell is functioning properly, then the body is staying homeostasis.
A cytotoxic T cell kills cells that have been infected by a virus or bacterium. It does this by puncturing the cell's membrane and by emitting a toxin that kills cells.
Yes, an increase in cholesterol can affect membrane permeability by making the cell membrane less fluid and more rigid, which can impact the movement of molecules in and out of the cell.
It selectively lets things transport into and out of the Cell.
CTX enters via Receptor mediated endocytosis (RME), also known as clathrin dependent endocytosis.
Neutrophils are active in phagocytizing (eating) bacteria and are present in large amount in the pus of wounds. But this bacteria makes a toxin which really causes the disease. The toxin is produced during cell growth, sporulation and lysis (break down). The white cells actually help to increase the toxin formation by eating them (lysis).Tetanus toxin is one of the three most poisonous substances known, the other two being the toxins of botulism and diphtheria. The vaccine is the most important form of prevention.
Flu viruses enter cells through endocytosis, a process where the cell engulfs the virus in a vesicle formed from the cell membrane. Diphtheria toxin enters cells through receptor-mediated endocytosis, where the toxin binds to a cell surface receptor and is internalized. Cholera toxin is taken up by clathrin-dependent endocytosis, where the toxin binds to a receptor on the cell surface and is internalized in clathrin-coated vesicles.
An environmental toxin that alters the role of the cytoskeleton is least likely to harm a plant. The cytoskeleton in plant cells is less complex compared to animal cells, making it less susceptible to disruption by toxins.
Toxins can get into cells through various mechanisms, such as binding to specific cell surface receptors and being taken up through endocytosis, penetrating the cell membrane directly, or being transported across the membrane by specific transporters. Once inside the cell, toxins can interfere with cell function and cause damage.