Erythrogenic
No. For something to be virulent it must be alive. Virulence usually refers to pathogens, like bacteria, viruses, and yeasts. But, pathogens, themselves, can create toxins by their metabolic activity. Still, however, the toxin itself wouldn't have the property of being virulent, only the "bug" would be.
The virulence factor list include capsules, fimbriae, outer membrane proteins, techoic acids, protein A, and m protein. Virulence factors are factors that allow pathogens to avoid host defense mechanisms and adversely affect the host.
Virulence is the degree of pathogenicity within a types of parasites. The factors of virulence are the colonization of a niche in the host, immunoevasion, immunosuppression, entry and exit of cells and obtaining nutrition from the host.
Enzymes can play a role in pathogenesis by helping pathogens invade host tissues, evade the immune system, and acquire nutrients. Toxins, on the other hand, cause damage to host cells or disrupt normal physiological processes to benefit the pathogen. Both enzymes and toxins are virulence factors that contribute to the pathogenicity of a microorganism.
Toxins
Toxins.
The virulence of bacteria depends on factors such as their ability to produce toxins, invade host tissues, and evade the host immune response. Bacteria that possess these virulence traits are often capable of causing severe infections and disease in the host, making them deadly. In contrast, bacteria lacking these virulence factors tend to be harmless and may even provide benefits to the host.
Toxins.
Some pathogens produce toxins.
Poisons produced by pathogenic bacteria are called "bacterial toxins."
Astrocytes are most responsible for preventing pathogens and toxins from entering the brain through blood.
Pore-forming toxins (PFTs) are a type of virulence factor produced by various pathogens, including bacteria, fungi, and some viruses. These toxins insert themselves into the cell membranes of host cells or competing microbes, creating pores that disrupt membrane integrity. This disruption leads to ion imbalance, loss of cellular function, and ultimately causes the targeted cell to burst and disintegrate, facilitating the spread of the pathogen.