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Elevated homocysteine levels are a prognostic indicator of arteriosclerosis and poor cardiovascular health.
Diets rich in B vitamins, such as folic acid, B6 and B12, tend to reduce serum homocysteine levels,
It is not clear whether lowering homocysteine levels in the cardiac patient will lead to improved outcomes, with respect to morbidity and mortality (especially as cardiovascular diseases tend to be multifactorial, often including a significant genetic component).
What else can I help you with?
When present in the blood levels in elevated levels the amino acid can damage arterial walls and increase the risk of coronary artery disease?
homocysteine
How do you control amount of homocysteine in the body?
Controlling homocysteine levels in the body primarily involves dietary adjustments and lifestyle changes. Consuming foods rich in B vitamins, particularly B6, B12, and folate, can help lower homocysteine levels. Regular physical activity, maintaining a healthy weight, and avoiding smoking can also contribute to balanced homocysteine levels. In some cases, healthcare providers may recommend supplements to help manage elevated homocysteine.
When present in the blood in elevated levels the amino acid can damage arterial walls and increase the risk of coronary artery disease?
homocysteine
Should homocysteines levels be high or low?
Homocysteine levels should generally be low, as elevated levels are associated with an increased risk of cardiovascular diseases, stroke, and other health issues. High homocysteine levels can indicate deficiencies in certain vitamins, such as B6, B12, and folate, which are essential for its metabolism. Maintaining healthy homocysteine levels through a balanced diet and appropriate supplementation can help reduce these health risks.
What is the amino acid homocysteine used for in the body?
Homocysteine is an intermediate in the metabolism of methionine, an essential amino acid. It can be converted back to methionine or further metabolized into cysteine or excreted from the body. Elevated levels of homocysteine are associated with an increased risk of cardiovascular disease.
What is true regarding B vitamins and homocysteine metabolism?
B vitamins, particularly B6, B12, and folate (B9), play a crucial role in homocysteine metabolism. They act as cofactors in enzymatic reactions that convert homocysteine into methionine or cysteine, thereby helping to regulate homocysteine levels in the body. Elevated homocysteine is associated with an increased risk of cardiovascular diseases, making adequate intake of these vitamins important for maintaining heart health. Deficiencies in any of these B vitamins can lead to increased homocysteine levels and related health issues.
What substance's levels are elevated when cardiac muscle tissue is damaged?
Troponin and CK MB
What amino acid intermediate causes arterial damage?
Homocysteine is an amino acid intermediate that can cause arterial damage. Elevated levels of homocysteine in the blood have been associated with an increased risk of developing cardiovascular diseases, including the development of atherosclerosis or the hardening and narrowing of the arteries. High homocysteine levels can damage the lining of blood vessels, promote blood clot formation, and contribute to inflammation, ultimately leading to arterial damage.
Which amino acid when present in the blood in elevated levels can damage arterial walls and increase the risk of coronary artery disease?
Homocysteine
What is homocysteine?
Homocysteine is an amino acid produced during the metabolism of methionine. High levels of homocysteine in the blood are associated with an increased risk of cardiovascular disease, stroke, and other health complications. Factors such as diet, genetics, and certain medical conditions can influence homocysteine levels.
High homocysteine levels associated with ischemia may be treated with which supplements?
High homocysteine levels may be treated with vitamin B6, B12 and folic acid.
Cardiac enzymes and troponin elevate what?
Cardiac enzymes and troponin levels do not elevate anything. Rather, when you have a heart attack (MI), they are elevated. Unlike damage to any other muscle in your body, damage to the cardiac muscle causes the release of specific cardiac enzymes. Therefore, if they are elevated, you had damage to your heart (i.e. heart attack). I disagree with the above: If both are elevated then you may have had a heart attack but, if cardiac enzymes alone are elevated and not troponin levels then you will not have had a heart attack. That's my personal experience. Troponin levels are now considered a more accurate indicator of damage to the heart muscle as a result of a heart attack than cardiac enzymes which can apply to any other muscle group rather than the heart. In fact to use the word 'cardiac' in this context is misleading because if you sprain your ankle for instance 'cardiac' enzymes will be released into the blood! So, where is the connection between damage to your ankle and that of your heart? Both enzymes therefore need to be elevated to indicate heart damage reliably.
