Chloride
Chloride ions can pass into the cell through voltage-gated chloride channels and ligand-gated chloride channels. These channels allow for the movement of chloride ions across the cell membrane in response to changes in voltage or binding of specific ligands.
Small chemicals called 'neurotransmitters' that are released into the synapse will diffuse across the synaptic cleft and bind to postsynaptic receptors (which may be on a cell body, or a dendrite). Examples of neurotransmitters are: adrenaline/epinephrine, acetylcholine, glutamate, serotonin, GABA, glycine and dopamine.
Neurotransmitters attach to specific proteins called receptors on the cell membrane. These receptors are typically ligand-gated ion channels or G protein-coupled receptors that initiate cellular responses when neurotransmitters bind to them.
When the sodium ions that entered the cell through the ion channels diffuse into the axon terminal of the neuron, they activate voltage-gated calcium ion channels. As calcium ions flow into the cell, neurotransmitters are released from the cell. These neurotransmitters diffuse across the synapse and activate sodium ion channels in the post-synaptic cell, allowing sodium to flow in and depolarize the cell enough to start another action potential.
Depends on what the pore does. If they are channels - they are called Channels. Voltage gated / ligand activated channels. If they are to communicate with neighbouring cells - they are called gap junctions.
Absence seizures are implicated with an abnormal imbalance of certain chemicals in the brain that modulate nerve cell activity (one of these neurotransmitters is called GABA, which functions as an inhibitor).
Excitatory signals, such as neurotransmitters like glutamate, can stimulate a neuron to transmit an electrical impulse. Inhibitory signals, like neurotransmitters GABA, can prevent a neuron from transmitting by hyperpolarizing the cell membrane and decreasing the likelihood of an action potential.
Ligand-gated channels are activated by binding of specific molecules (ligands) to the channel, while voltage-gated channels are activated by changes in the electrical potential across the cell membrane. Ligand-gated channels are regulated by the presence of ligands, while voltage-gated channels are regulated by changes in membrane potential.
Ligand-gated channels are activated by binding of specific molecules (ligands) to the channel, while voltage-gated channels are activated by changes in the electrical potential across the cell membrane. Ligand-gated channels are regulated by the presence of ligands, while voltage-gated channels are regulated by changes in membrane potential.
Ligand-gated channels are activated by binding of specific molecules (ligands) to the channel, while voltage-gated channels are activated by changes in the electrical potential across the cell membrane. Ligand-gated channels are regulated by the presence of ligands, while voltage-gated channels are regulated by changes in membrane potential.
Ligand-gated ion channels are activated by binding of specific molecules (ligands) to the channel, while voltage-gated ion channels are activated by changes in the electrical potential across the cell membrane. Ligand-gated channels are regulated by the presence of ligands, while voltage-gated channels are regulated by changes in membrane potential.
Ligand-gated ion channels are activated by binding of specific molecules (ligands) to the channel, while voltage-gated ion channels are activated by changes in the electrical potential across the cell membrane. Ligand-gated channels are regulated by the presence of ligands, while voltage-gated channels are regulated by changes in membrane potential.