Vaccines are substances that activate the immune system against specific pathogens without causing illness. They typically contain weakened or inactivated parts of the pathogen, such as proteins or sugars, or genetic material that instructs cells to produce a harmless piece of the pathogen. This exposure trains the immune system to recognize and respond more effectively if the body encounters the actual pathogen in the future.
When a lymphocyte recognizes a pathogen, it first binds to specific antigens on the pathogen's surface through its unique receptors. This recognition activates the lymphocyte, leading to its proliferation and differentiation into effector cells, such as cytotoxic T cells or antibody-secreting B cells. These effector cells work to eliminate the pathogen, either by directly killing infected cells or by producing antibodies that neutralize the pathogen. Additionally, memory cells are formed to provide long-term immunity against future infections by the same pathogen.
During a second infection, the immune system quickly activates memory cells that were generated during the first exposure to the pathogen. These memory T and B cells recognize the specific antigens of the pathogen and mount a faster and more robust immune response. This accelerated response can lead to quicker clearance of the pathogen and often results in milder symptoms or even complete prevention of illness. This phenomenon is the basis for how vaccines work, training the immune system to respond effectively to future infections.
each pathogen has a antigene which the antibodies attach to so each pathogen needs a unique antibodie
Toll-like receptors are proteins that recognize specific molecules on pathogens, like bacteria or viruses. When a toll-like receptor detects a pathogen, it triggers a signaling cascade that activates the immune response, leading to the elimination of the pathogen. This helps the immune system to quickly detect and respond to potential threats.
For an infection to occur, a susceptible host must be present, meaning that the host's immune system is either compromised or not fully equipped to combat the pathogen. This often includes individuals with weakened immune responses, such as those with chronic illnesses, the elderly, or young children. Additionally, the host must have specific receptors or cellular environments that allow the pathogen to attach, invade, and replicate. Without these conditions, the pathogen is unlikely to establish an infection.
N/aA pathogen by definition is a disease causing organism
The substance introduced into the body to stimulate the production of chemicals that destroy specific disease-causing viruses and organisms is called a vaccine. Vaccines work by exposing the immune system to a harmless component of the pathogen, prompting the body to generate an immune response and "memory" cells. This prepares the immune system to recognize and combat the actual pathogen if encountered in the future, thereby providing immunity.
You'd most likely want to expose yourself to a "dead" version of that pathogen via vaccination.
The lungs are the primary site of infection for many respiratory illnesses, such as pneumonia or tuberculosis. However, infections can also occur in other parts of the body, such as the skin, urinary tract, or gastrointestinal tract. The specific location of the infection depends on the type of pathogen involved.
the inactive form still has the antigens (protein markers) specific to that pathogen on the surface. the immune system develops specific responses to this pathogen when it is encountered after a vaccination. as it is inactive the person does not suffer the effects of this pathogen, but when an active form is encountered later the immune system is quicker to respond due to the fact that it now recognises those antigens.
That is the job of the B-cells, or B Lymphocytes.
it causes other diseases