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Having too many or too few normal genes can disrupt the delicate balance of gene expression and regulation necessary for proper cellular function. This imbalance can lead to overproduction or underproduction of proteins, affecting vital biological processes and pathways. For instance, an excess of certain proteins may lead to uncontrolled cell growth, while a deficiency can result in insufficient maintenance of cellular functions, both of which can contribute to the development of various disorders. Ultimately, the precise dosage of genes is crucial for maintaining health and homeostasis.
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What are defective genes?
Defective genes are genes that have mutations or alterations that can lead to abnormal function or health conditions in individuals. These mutations can disrupt the normal processes controlled by the gene, potentially resulting in genetic disorders or diseases.
What is an application of the human genome project that involves the insertion of normal genes int o cells with defective genes in an attemp to correcy genetic disorders?
Gene Therapy
What involves replacing faulty genes with normal working genes in order to provide a cure for a genetic disorder?
Gene therapy involves replacing faulty genes with normal working genes to provide a cure for a genetic disorder. This can be done by inserting the normal gene into the patient's cells using a vector, such as a virus, to deliver the gene safely. Gene therapy holds promise for treating a wide range of genetic disorders.
Sex linked disorders are caused by males' having?
Sex-linked disorders are caused by genes located on the sex chromosomes, particularly the X chromosome. Since males have only one X chromosome, mutations in genes on this chromosome are more likely to be expressed in males compared to females who have two X chromosomes. This is why sex-linked disorders are more commonly seen in males.
What are two sex linked disorders?
Genes and phenotype
Do you choose to have disorders?
No that is passed through something called Genes.
Why do you think having too many or too few normal genes creates disorder?
Having too many or too few normal genes can disrupt the delicate balance of gene expression and regulation necessary for proper cellular function. An excess of certain genes can lead to overproduction of proteins, causing cellular overload and potential toxicity, while a deficiency can result in insufficient protein levels needed for critical biological processes. This imbalance can contribute to developmental disorders, metabolic issues, and increased susceptibility to diseases. Ultimately, the precise regulation of gene dosage is crucial for maintaining health and homeostasis in living organisms.
Why can females but not males carriers of sex linked genetic disorders?
Several genetic disorders are caused by genes on the X chromosomes.
What are genetic disorders that involve more than one defective gene?
Some examples of genetic disorders involving more than one defective gene are cystic fibrosis, which is caused by mutations in the CFTR gene and other modifier genes; and certain types of neurofibromatosis, which result from mutations in both the NF1 and NF2 genes. These disorders demonstrate the complexity of genetic inheritance and the interactions between multiple genes.
What do mutations in genes cause?
Mutations in genes can cause changes in the structure or function of the corresponding proteins, leading to various outcomes such as genetic disorders, cancer, or altered traits. Mutations can disrupt normal cellular processes, affect gene regulation, or result in the production of abnormal proteins.
Why can females but not males be carriers of sex linked genetic disorders?
Females have two X chromosomes, allowing them to be carriers of sex-linked genetic disorders on one X chromosome while having a normal allele on the other X chromosome. Males have only one X chromosome and one Y chromosome, so any genetic disorder on their single X chromosome will manifest as the disorder rather than being masked.
What are some examples of mental disorders that are caused by X-linked genes?
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